1964
DOI: 10.1172/jci104980
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Relationship between Plasma Triglycerides and Removal of Chylomicrons*

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Cited by 242 publications
(58 citation statements)
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“…Figure 3C illustrates that HDL cholesteryl ester as percentage of plasma declined after an oral fat load from 10% (6-11) to 8% (4-11), p<0.05, analyzed from 0 to 8 hours. LDL and TRL cholesteryl ester did not change significantly from 8 1 % (78-88) to 82% (79-88), and from 10% (5-12) to 11% (5)(6)(7)(8)(9)(10)(11)(12)(13)(14), respectively.…”
Section: Cholesteryi Ester Distribution Between Upoprotelnsmentioning
confidence: 84%
“…Figure 3C illustrates that HDL cholesteryl ester as percentage of plasma declined after an oral fat load from 10% (6-11) to 8% (4-11), p<0.05, analyzed from 0 to 8 hours. LDL and TRL cholesteryl ester did not change significantly from 8 1 % (78-88) to 82% (79-88), and from 10% (5-12) to 11% (5)(6)(7)(8)(9)(10)(11)(12)(13)(14), respectively.…”
Section: Cholesteryi Ester Distribution Between Upoprotelnsmentioning
confidence: 84%
“…For example, higher plasma VLDL triglyceride levels are associated with lower plasma HDL-C levels, 531 smaller, denser LDL particles, 5 -32 and increased postprandial accumu- lation of triglyceride-rich particles of intestinal origin. 33 All of these effects may enhance the atherogenicity of circulating lipoproteins and increase coronary disease risk in men compared with women. 313234 The differences in lipoprotein pattern between men and women (higher triglycerides, lower HDL-C, and smaller, denser LDL particles) are paralleled by the differences in lipoprotein pattern between people with non-insulin-dependent diabetes (NIDDM) and people with normal glucose tolerance.…”
Section: Discussionmentioning
confidence: 99%
“…A brief summary of all studies published after 1987 is shown in Table 3. Studies before 1987, which in fact deal almost exclusively with those published before 1964, are not included as none of them took into account the fact that the level of fasting plasma triglycerides is such an important determinant of the evolution of postprandial plasma lipids after fat intake [109]. and B-100 Postprandial elevation of TRL ApoB-100 seems specifically associated with CAD + higher plasma triglycerides in the late postprandial phase or a generally elevated triglyceride curve (area under curve measurement) even after correction for the fasting level.…”
Section: Clinical Evidence For a Link Between Disturbed Metabolism Ofmentioning
confidence: 99%