Recurrent Transient Monocular Blindness with Ophthalmic Artery Stenosis

Park, Man‐Seok; Kim, Joon Tae; Lee, Kee Ra; Lee, Seung-Han; Choi, Seong Min; Kim, Byeong C.; Kim, Myeong Kyu; Cho, Ki-Hyun

doi:10.1159/000114047

Cited by 6 publications

(4 citation statements)

References 6 publications

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“… 33 Cerebral angiography may show focal stenosis at the origin of the central retinal artery or ophthalmic artery. 34 Viewing the fundus during symptomatic episodes in these cases reveals arteriolar constriction without emboli, and TVL can occur with bending the head down, suggesting hemodynamic change rather than emboli. Treatment with aspirin or calcium channel blockers decreases symptoms, possibly by reducing platelet activation and vasospasm.…”

Section: Tvl and Thromboembolic Vascular Diseasementioning

confidence: 86%

Update on the evaluation of transient vision loss

Pula¹,

Kwan²,

Yuen³

et al. 2016

OPTH

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Transient vision loss may indicate underlying vascular disease, including carotid occlusion and thromboembolism, or it may have a more benign etiology, such as migraine or vasospasm. This review focuses on the differential diagnosis and workup of patients presenting with transient vision loss, focusing on several key areas: the relationship to thromboembolic vascular disease, hypercoagulable testing, retinal migraine, and bilateral vision loss. The objective is to provide the ophthalmologist with information on how to best manage these patients. Thromboembolic etiologies for transient vision loss are sometimes managed with medications, but when carotid surgery is indicated, earlier intervention may prevent future stroke. This need for early treatment places the ophthalmologist in the important role of expediting the management process. Hospital admission is recommended in patients presenting with transient symptoms within 72 hours who meet certain high-risk criteria. When the cause is giant cell arteritis, ocular ischemic syndrome, or a cardioembolic source, early management of the underlying condition is equally important. For nonthromboembolic causes of transient vision loss such as retinal migraine or retinal vasospasm, the ophthalmologist can provide reassurance as well as potentially give medications to decrease the frequency of vision loss episodes.

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Section: Tvl and Thromboembolic Vascular Diseasementioning

confidence: 86%

Update on the evaluation of transient vision loss

Pula¹,

Kwan²,

Yuen³

et al. 2016

OPTH

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“…7 Transcranial Doppler ultrasound and cerebral angiography are necessary to confirm the diagnosis, and antiplatelet therapy may be effective in preventing recurrent attacks, regardless of their mechanism. 16 Similar episodes of TVL can also occur with ophthalmic artery occlusion. 17 Cerebral angiography is required to confirm the diagnosis and antiplatelet therapy is often effective in preventing recurrences.…”

Section: Ophthalmic Artery Stenosis and Occlusionmentioning

confidence: 96%

“…Stenosis of the ophthalmic artery due to atheroma can produce isolated episodes of transient monocular visual loss, as a consequence of retinal and optic nerve hypoperfusion 16 or embolism to the retinal www.internat-ophthalmology.com arteries. 7 Transcranial Doppler ultrasound and cerebral angiography are necessary to confirm the diagnosis, and antiplatelet therapy may be effective in preventing recurrent attacks, regardless of their mechanism.…”

Section: Ophthalmic Artery Stenosis and Occlusionmentioning

confidence: 99%

Transient Visual Loss

Thurtell¹,

Rucker²

2009

International Ophthalmology Clinics

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“…the origin of the central retinal artery or ophthalmic artery. 8 Stenosis of the ophthalmic artery is very rare. According to a previous study, the prevalence of ophthalmic artery stenosis is approximately 2% in patients suffering from amaurosis fugax.…”

Section: Cerebral Angiography May Show Focal Stenosis Atmentioning

confidence: 99%

Ipsilateral Ophthalmic Artery Stenosis in Amaurosis Fugax: A Case Report

Agustiawan¹,

Iskandar²,

Mokoagow³

et al. 2021

IJRetina

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Introduction: Amaurosis fugax is caused by an abrupt reduction of blood flow to the retina. Determining the etiology of amaurosis fugax should ensure proper management. Case Report: A 47-year-old female patient who had the first episode of amaurosis fugax in her right eye was referred to our hospital. The amaurosis fugax resolved spontaneously, however, we found a 3 mm long stenosis at her right ophthalmic artery during magnetic resonance angiography. She had clinical histories of untreated hypertension and dyslipidemia. Transient ischemic attack (TIA) was suspected and unfractionated heparin, aspirin, antihypertensive agent, and statin were given. Treatments were maintained, the symptoms had not recurred in the following 6 months after the event. Discussion: Stenosis of the ophthalmic artery is very rare. It occurs in approximately 2% of patient suffering from amaurosis fugax. In our case, stenosis of the right ophthalmic artery due to thromboembolism might cause retinal ischemia leading to a transient visual loss. Conclusion: This case suggests stenosis of ophthalmic artery as the cause of amaurosis fugax. Keywords: amaurosis fugax, ipsilateral, ophthalmic artery, stenosis

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Recurrent Transient Monocular Blindness with Ophthalmic Artery Stenosis

Cited by 6 publications

References 6 publications

Update on the evaluation of transient vision loss

Update on the evaluation of transient vision loss

Transient Visual Loss

Ipsilateral Ophthalmic Artery Stenosis in Amaurosis Fugax: A Case Report

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