“…The pathophysiology of spinal myoclonus seems to involve abnormal hyperactivity of the local interneurons of the dorsal horn with loss or impairment of inhibition of suprasegmental descending pathways [ 5 , 6 ]. Furthermore, several case reports of spinal myoclonus subsequent to neuraxial anesthesia have been published [ 8 , 9 , 10 , 11 , 12 ]. According to these previous reports, the onset, duration, and recurrence of spinal myoclonus are not predictable and are not related to dose or baricity of local anesthetics or concomitant drugs used in spinal anesthesia.…”