Recurred Sphenoethmoidal Mucocele After Treated With Simple Endoscopic Marsupialization

Abstract

mucosal tissue and cartilage tissue to reconstruct the palpebral conjunctiva and tarsus (Fig. 2C). Suture the mucosal which was faced inward cartilage complex to the defect of left lower eyelid, mucosa was faced inward (Fig. 2D). Fully separate the subcutaneous tissue along the outside of the left lower eyelid to form a flap to cover the inner defect wound. When there was no obvious tension, the mucosa of the mucosal cartilage complex was sutured to the edge of the flap to reconstruct the eyelid margin. Then the subcutaneous tissue and skin of the skin flap were sutured (Fig. 2E). DISCUSSIONOptimal restoration of esthetics and anatomical function, with a minimum of surgical complication is the ultimate goal of eyelid reconstruction. It is essential to pay attention to reconstitution of the eyelid structure. At the anterior layer, beautiful skin needs to be created, and the function of the orbicularis muscle must be maintained as much as possible. Creating a posterior lamella replacing tarsus at the eyelid margin and providing for a smooth mucosal surface that protects and preserves the cornea. 3 In conclusion, the author used buccal mucosa to repair the conjunctival surface of the lower eyelid defect maintain the function of the conjunctival surface, reduce the damage to the original ocular surface environment. Using autologous auricular cartilage as the skeleton of the eyelid defect, provided better support for the flap, and the site had a good esthetic appearance after surgery. Using skin flap to repair the skin surface of the lower eyelid defect, the eyelid defect can be repaired and good clinical results can be obtained. What we are most worried about is the cartilage did not survive and scar contracture causing lower eyelid ectropion. Fortunately, during the follow-up of this case (Fig. 2F), no related complications were found.

Secondary optic neuropathy due to sphenoid-ethmoidal sinus mucocele

“…The most prevalent intracranial pathologies that can lead to optic neuropathies are lesions in the sellar and parasellar areas such as pituitary tumors, meningiomas, craniopharyngiomas, and intracerebral lesions affecting the visual pathways, as well as primary or secondary hydrocephalus. In addition to numerous main ophthalmologic illnesses [3], rhinogenic optic neuritis may also be induced by sphenoid sinus mucocele with sinusitis, ethmoiditis, and onodi cell inflammation [4][5][6][7].…”