Abstract:Background
Previous research has shown that high levels of trait social anxiety (SA) disrupt the social repair processes following a painful social exclusion, but the cognitive mechanisms involved in these processes and how trait SA may disrupt them remain unknown.
Methods
We conducted a preregistered study on Prolific participants (
N
= 452) who were assigned to experience either social exclusion or inclusion and were then exposed to follow-u… Show more
“…Furthermore, clinical examinations showed that individuals with SAD lacked motivation towards social rewards 108 . If the predicted reward value from a positive social outcome is substantially smaller compared to the potential social loss, individuals may choose avoidance behaviours 109,110 . Thus, our third factor (a pair) was altered social reward and loss processing, encoded in SocGain and SocLoss respectively.…”
Understanding the mechanisms underlying psychopathology is a central goal in clinical science. While existing theories have demonstrated high clinical utility, they have provided limited quantitative and experimentally falsifiable explanations of psychopathological mechanisms. Previous computational modelling studies have primarily focused on isolated factors, posing challenges for advancing clinical theories. To address this gap and leverage the strengths of clinical theories and computational modelling in a synergetic manner, it is crucial to develop a quantitative model that integrates major vulnerability factors within a comprehensive psychopathological model. In this study, using social anxiety disorder (SAD) as an example, we present a novel approach by combining active inference modelling, an innovative computational approach that elucidates human cognition and action, with cognitive-behavioural theory (CBT), a well-established clinical framework. This CBT-informed quantitative account integrates multiple vulnerability factors to elucidate the psychopathology of SAD. Through a series of simulations, we systematically delineate their effects on pathological behaviours. This resultant model inherits the conceptual comprehensiveness of CBT and the quantitative rigor of active inference modelling, revealing previously elusive pathogenetic pathways and enabling the formulation of specific predictions for empirical studies. Furthermore, this resultant model provides a strong theoretical foundation for precision medicine in SAD by allowing for individual differences in the symptom development trajectory. Overall, this research presents the first quantitative model of SAD that unifies major pathogenesis factors proposed by CBT. It highlights the feasibility and potential of integrating clinical theory and computational modelling to advance our understanding of psychopathology.
“…Furthermore, clinical examinations showed that individuals with SAD lacked motivation towards social rewards 108 . If the predicted reward value from a positive social outcome is substantially smaller compared to the potential social loss, individuals may choose avoidance behaviours 109,110 . Thus, our third factor (a pair) was altered social reward and loss processing, encoded in SocGain and SocLoss respectively.…”
Understanding the mechanisms underlying psychopathology is a central goal in clinical science. While existing theories have demonstrated high clinical utility, they have provided limited quantitative and experimentally falsifiable explanations of psychopathological mechanisms. Previous computational modelling studies have primarily focused on isolated factors, posing challenges for advancing clinical theories. To address this gap and leverage the strengths of clinical theories and computational modelling in a synergetic manner, it is crucial to develop a quantitative model that integrates major vulnerability factors within a comprehensive psychopathological model. In this study, using social anxiety disorder (SAD) as an example, we present a novel approach by combining active inference modelling, an innovative computational approach that elucidates human cognition and action, with cognitive-behavioural theory (CBT), a well-established clinical framework. This CBT-informed quantitative account integrates multiple vulnerability factors to elucidate the psychopathology of SAD. Through a series of simulations, we systematically delineate their effects on pathological behaviours. This resultant model inherits the conceptual comprehensiveness of CBT and the quantitative rigor of active inference modelling, revealing previously elusive pathogenetic pathways and enabling the formulation of specific predictions for empirical studies. Furthermore, this resultant model provides a strong theoretical foundation for precision medicine in SAD by allowing for individual differences in the symptom development trajectory. Overall, this research presents the first quantitative model of SAD that unifies major pathogenesis factors proposed by CBT. It highlights the feasibility and potential of integrating clinical theory and computational modelling to advance our understanding of psychopathology.
“…Fung and Alden [30] show that the intensity of social pain after the initial social exclusion could mediate the anxiety level of participants who experience subsequent social interaction. For individuals with higher social anxiety, their social repair is less than average social anxiety level individual after social exclusion, suggesting they might be less functional to release the negative effect of social pain, have a higher possibility to carry more weight of their past painful social event [31]. These effects could elevate and maintain the higher level of social anxiety and keep susceptible individuals more vulnerable within social situations.…”
Section: Higher Social Anxiety Inhibits the Need Repairmentioning
confidence: 99%
“…They tend to protect themselves by socially withdrawing [37]. Hudd [38] explained that the reaction of high social anxiety individuals might be because they are more sensitive to threats (e.g., excessive attention and responsivity to threat signals), and this overactive threat-avoidance motivation system could drive their behaviour within interpersonal situations. Besides, there are fewer rewards for high social anxiety individuals to reconnect to others.…”
Section: Higher Social Anxiety Inhibits the Need Repairmentioning
Social exclusion, or ostracism, is being excluded or rejected by others. It is the common behaviour within social situations. Detecting social exclusion could be essential for the survival of human creatures, as social exclusion could threaten the basic needs of human creatures: self-esteem and feelings of belongingness. Williams temporal need threat model introduces how individuals react toward ostracism. Usually, individuals can recover from social exclusion quickly by behaving pro-socially to reconnect to others and fortify the basic need(s) threatened by social exclusion. However, for individuals with higher social anxiety or social anxiety disorder, the negative social belief about themselves (unpopular, shyness) within a social situation inhibits their recovery speed from exclusion and desire to reconnect to others. When social exclusion happens, individuals with higher social anxiety or social anxiety disorder become more vulnerable in social situations. Individuals lose their ability to get used to social situations when they suffer long-term social exclusion. Acceptance and Commitment Therapy is suggested to be a helpful therapy for individuals suffering from long-term social exclusion and social anxiety disorder.
“…Evaluations on coping themes in pregnant women have concluded that quarantine has strengthened social support networks for pregnant women as the family members are at home and dedicate time and consideration to them, which has contributed to less threat-avoidance behaviors and thus moderately decelerated anxiety among pregnant women. On the contrary, with the lockdown policies in place, hampering pregnant women's access to essential obstetric care services accompanied by less involvement in social events has led to a significant decline in applying reward-seeking coping strategies and thus a substantial rise in major depression incurred [26,27].…”
Section: The Impact Of Engagement In Reward-seeking and Threat-avoida...mentioning
The COVID-19 pandemic has struck nations worldwide, pushing worldwide health and socioeconomic systems to extreme limits. Various factors, such as drastic alterations in public environments, prolonged quarantine, revenue loss, and anxiety of disease contraction, have caused mental turmoil. Although there was a need to cope with an excess of psychological strain among the public, post-COVID patients, and those with a previously diagnosed psychiatric condition, mental health programs faced a substantial decline in services, mirroring the dramatic rise in psychological issues. Interestingly, certain coping strategies play protective or deleterious effects on mental health outcomes. Moreover, social media exposure has played a double-edged role in the mental health of the public during the pandemic, leaving grounds for further debates. Protean cultural themes have taken center stage in the discussion on social resilience and compliance to COVID-19 measures, driving their impact through certain work ethics, social capital, and public attitudes in different societies. On the other hand, exceedingly rising poverty rates cemented the deleterious economic impact of the pandemic. Attention has been called to the racial implications of the pandemic, driving millions of individuals with low socioeconomic position (SEP) and belonging to minority groups out of the paid workforce. Interestingly, we turn attention to an array of elements implicated in this dramatic effect, such as public transport, living arrangements, and health insurance coverage among these vulnerable groups. We attempt to address the mechanisms COVID-19 channeled its mental health and socioeconomic impacts by explaining the risk factors and pave the way for stronger crisis management in the future by evaluating the socioeconomic and psychological effects in stark detail.
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