Re-evaluation of the atrial contribution to ventricular function

Kosowsky, Bernard D.; Scherlag, Benjamin J.; Damato, Anthony N.

doi:10.1016/0002-9149(68)90284-1

Cited by 113 publications

(30 citation statements)

References 13 publications

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“…7) show that with the PR preserved, institution of ectopic right ventricular activation produced only minor changes in LV parameters. This would agree generally with studies previously reported indicating variable low magnitude deleterious effects of ectopic ventricular activation per se (18)(19)(20)(21).…”

Section: Discussionsupporting

confidence: 93%

Left atrial booster function in valvular heart disease

Heidenreich¹,

Shaver²,

Thompson³

et al. 1970

J. Clin. Invest.

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Section: Discussionsupporting

confidence: 93%

Left atrial booster function in valvular heart disease

Heidenreich¹,

Shaver²,

Thompson³

et al. 1970

J. Clin. Invest.

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“…Studies by Abildskov and co-workers (1965), as well as Kosowsky et al (1968), demonstrated that abnormal depolarization, as a result of ventricular pacing, was associated with small changes in left ventricular function. The changes included a decrease in both peak systolic pressure and the rate of rise of pressure.…”

Section: Discussionmentioning

confidence: 97%

“…Aberrant ventricular activation associated with an asynchronous ventricular contraction has been evaluated by various investigators (Abildskov et al .. 1965;Finney, 1965;Gilmore et al .. 1963;Kosowsky et al .. 1968;Samet et al .. 1966). Studies by Abildskov and co-workers (1965), as well as Kosowsky et al (1968), demonstrated that abnormal depolarization, as a result of ventricular pacing, was associated with small changes in left ventricular function.…”

Section: Discussionmentioning

confidence: 99%

Preload reduction with right ventricular pacing: Effects on left ventricular hemodynamics and contractile pattern

Hamby

Aintablian

1980

Clinical Cardiology

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Summary: Studies were performed in 32 patients to evaluate left ventricular pressure-volume changes and contractile pattern during right ventricular pacing as compared to normal sinus rhythm. -Coronary artery disease was present in 27 patients, while 5 patients (control group) had no evidence of coronary artery or left ventricular disease. Studies were performed during both normal sinus rhythm and right ventricular pacing at comparable heart rates, utilizing angiographic methods to determine heart volumes. Right ventricular pacing in aU patients resulted in decreased left ventricular systolic (p< 0.01) and diastolic (p< 0.01) pressures and decreased stroke work (p< 0.00 I). In the control group, right ventricular pacing caused a decrease in left ventricular end-diastolic volume (p< 0.0\) and stroke volume (p< 0.0\), with no change in ejection fraction. The patients with coronary artery disease were divided into four groups, dependent on the left ventricular contractile pattern during normal sinus rhythm and the percentage of change in hemiaxis shortening during right ventricular pacing. In group A (six patients with asynergy) and group B (seven patients with asynergy), there was no significant change in the percentage of hernia xis shortening during right ventricular pacing when compared to normal sinus rhythm. Ventricular volume studies in these patients (groups A and B) were similar to the control groups and no change in contractile pattern was observed during pacing. In group C, twelve patients had asynergy and a 10% increase in percentage of hemiaxis shortening during right ventricular pacing when compared to normal sinus rhythm. Right ventricular pacing resulted in decreased end-diastolic pressure (p< 0.0 I) and end-diastolic volume (p< 0.001), no change in stroke volume, and an increased ejection fraction (p< 0.01). Contractile patterns improved in aU patients in group C during pacing. Group D consisted of two patients with asynergy and a 10% decrease in percentage of hernia xis shortening during pacing, associated with a decrease in end-diastolic volume and ejection fraction with deterioration of left ventricular contractile pattern.These results indicate that right ventricular pacing in patients with coronary artery disease decreases preload, which may be accompanied by improved left ventricular contractile pattern (11/27) and in some patients (2/27) deterioration of left ventricular function.

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“…10,11 The QRS with selective capture will be essentially identical to native conduction, whereas nonselective QRS morphology will have a characteristic pseudo-delta wave akin to pre-excitation associated with an anteroseptal pathway. If the His pacing site is subvalvular, His capture is more likely to be encountered at higher outputs (resulting in non-selective His bundle capture, described elsewhere 4,9,12 and below), with lower output resulting in ventricular capture only prior to loss of capture (Video 2 and Figure 4). This pacing site is the one most often chosen when para-Hisian pacing maneuvers are used to characterize the presence or absence of retrograde-conducting accessory pathways.…”

Section: Methodsmentioning

confidence: 99%

“…However, by definition, the most physiological approach to activating the ventricles with external electrical stimuli is via the His bundle and through the His-Purkinje system. His bundle pacing was first demonstrated experimentally almost 50 years ago, 4 and was subsequently shown to be feasible clinically as a permanent clinical pacing strategy 16 years ago. 5 More recently, with the advent of site-selective lead delivery systems, His bundle pacing has been garnering increased interest, with recent publications demonstrating feasibility and clinical utility in the settings of patients with and without intrinsic His-Purkinje disease.…”

Section: Introductionmentioning

confidence: 99%

Step-wise Approach to Permanent His Bundle Pacing

Lustgarten¹

2016

JICRM

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ABSTRACT. There has been a recent upsurge of interest in permanent His bundle pacing, given increased understanding and appreciation of the role dyssynchronous ventricular activation plays in congestive heart failure and atrial fibrillation. Permanent His bundle pacing gives implanters the ability to avoid causing ventricular dyssynchrony in patients dependent on ventricular pacing, and can provide an alternative means to implementing cardiac resynchronization therapy in patients with bundle branch disease and congestive heart failure. This paper describes a step-wise approach to implanting permanent His bundle pacing leads, with a clear demonstration of techniques and observations encountered during the course of typical His bundle lead implantations, including consideration of potential pitfalls and workarounds.

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Re-evaluation of the atrial contribution to ventricular function

Cited by 113 publications

References 13 publications

Left atrial booster function in valvular heart disease

Left atrial booster function in valvular heart disease

Preload reduction with right ventricular pacing: Effects on left ventricular hemodynamics and contractile pattern

Step-wise Approach to Permanent His Bundle Pacing

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