Radiometric determination of uranium accumulated in bacterial cells

Chwistek, M.; Chmielowski, Jerzy; Tomza, I.

doi:10.1016/0039-9140(88)80070-5

Cited by 4 publications

(2 citation statements)

References 7 publications

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“…While some UO: ' probably binds to anionic membrane sites throughout the tubules (path C), greatest deposition may be in the distal third of the proximal tubules (Bowman and Foulkes 1970;Luessenhop et al 1958;Haley 1982). The specific membrane components that bind UO: ' are not known, but the potential importance of phosphate groups, particularly those of phospholipids, as binding sites for U has been noted in many studies for a variety of cell types (Bernheim 197 1 ;Hu 1972;Zeiske 1978;Bulman 1980;Friis and MyersKeith 1986;Pasquale et al 1986;Chwistek et al 1988). According to Hu (1972), the affinity of UO: ' for other major ligands in cell membranes is small compared to its affinity for phosphate groups, particularly at low concentration of UO: ', and for this reason UO," has been used as a chemical reagent specifically for phosphate groups of phospholipids.…”

Section: Behavior Offiltered U In the Renal Tubulementioning

confidence: 96%

The Behavior and Chemical Toxicity of U in the Kidney

1989

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By the late 1950s, the views had evolved that 1) U does not enter cells but damages the kidneys by binding to the luminal membranes of renal tubular cells, interfering with reabsorption of glucose, sodium, amino acids, protein, water, and other substances, and causing slow cell death by suppression of respiration; and 2) U does not cause significant damage to the kidneys at concentrations below 3 micrograms U g-1 kidney. Although there has not been a major unified effort in the past three decades to update the toxicology of U as a nephrotoxin, there have been numerous isolated studies that may be useful in reevaluating these longstanding views on the behavior and action of U in the kidneys and the renal U concentration at which toxic effects may become evident. This paper is a brief review and synthesis of current information on U nephrotoxicity. Much more experimental research and reevaluation of existing data are needed concerning U nephrotoxicity, particularly for the case of chronic exposure.

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Section: Behavior Offiltered U In the Renal Tubulementioning

confidence: 96%

The Behavior and Chemical Toxicity of U in the Kidney

1989

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“…Соединения германия могут накапливаться и поглощаться некоторыми бактериями. В работах [27,28] биоакку-муляция германия исследована как модель неспецифического поглощения некоторых металлов микроорганизмами, в частности бактериями Pseudomonas putida. В 20-е годы прошлого века появились первые со-общения о стимуляции образования крас-ных кровяных телец в результате введения людям и животным нетоксичных доз диок-сида германия [4].…”

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Some bioactive coatings of implants (literature review)

Nazarov¹,

Kuzmanin²

2016

I.P. Pavlov Russian Medical Biological Herald

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О НЕКОТОРЫХ БИОАКТИВНЫХ ПОКРЫТИЯХ ИМПЛАНТАТОВ (ОБЗОР ЛИТЕРАТУРЫ) Е.А. Назаров, С.А. КузьманинРязанский государственный медицинский университет им. акад. И.П. Павлова, г. Рязань В статье рассмотрены возможности улучшения свойств покрытий имплантатов путѐм введении в их состав германиевых и алмазоподобных углеродных компонен-тов. Проанализированы биологические свойства неорганического германия и его соединений. Отмечено, что германий в составе кальций-фосфатных покрытий им-плантатов положительно влияет на процессы остеоинтеграции.Ключевые слова: имплантат, покрытие.

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Preconcentration and determination of promethazine at lipid‐modified carbon paste electrodes

et al. 1989

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Lipid-modified carbon paste electrodes were electrochemically characterized and their potential for drug analysis was compared with conventional carbon paste electrodes. The presence of lipids (phospholipids, fatty acids) in the paste matrix provided enhanced current responses with improved reproducibility. With 5 miputes of accumulation, phenothiazines exhibit a 40-fold enhancement of the response compared to that obtained without accumulation. As a result, a detection limit of 1 X attained. The response was characterized with respect to pH, preconcentraiion potential, accumulation time, paste composition, possible interferences, and other variables. The determination of the tranquilizer in serum and urine required no preliminary treatment other than dilution with phosphate buffer and medium exchange, Detection limits are 5 x M was ' M in urine and 2 X lo-' M in serum.

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Radiometric determination of uranium accumulated in bacterial cells

Cited by 4 publications

References 7 publications

The Behavior and Chemical Toxicity of U in the Kidney

The Behavior and Chemical Toxicity of U in the Kidney

Some bioactive coatings of implants (literature review)

Preconcentration and determination of promethazine at lipid‐modified carbon paste electrodes

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