Radiological intervention in budd-chiari syndrome: Techniques and outcome in 18 patients

Griffith, J. F.; Mahmoud, Ahmed A.; Cooper, Stanley G.; Elias, Elwyn; West, Roy; Olliff, Simon

doi:10.1016/s0009-9260(96)80005-5

Cited by 83 publications

(54 citation statements)

References 26 publications

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“…Acute thrombotic events require anticoagulation with heparin. Thrombolytic therapy 77,78 or radiologic intervention 79 should be strongly considered in patients with acute onset of Budd-Chiari syndrome. Thrombocytopenia often complicates PNH, and this issue must be addressed when formulating an anticoagulation management plan.…”

Section: Thrombosismentioning

confidence: 99%

Diagnosis and management of paroxysmal nocturnal hemoglobinuria

Parker¹

2005

Blood

668

740

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Section: Thrombosismentioning

confidence: 99%

Diagnosis and management of paroxysmal nocturnal hemoglobinuria

Parker¹

2005

Blood

668

740

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“…Systemic thrombolytic therapy, or thrombolytic therapy delivered via canalization directly to the affected site, can be considered in patients with acute onset of Budd-Chiari syndrome. [48][49][50][51] If there is no contraindication, anticoagulation should continue indefinitely for a patient with PNH (even if that patient is treated with eculizumab) who has experienced a thromboembolic complication. For PNH patients on long-term anticoagulation, a vitamin K antagonist such as Coumadin has been used customarily.…”

Section: How I Treat Pnh Based On Disease Classificationmentioning

confidence: 99%

Update on the diagnosis and management of paroxysmal nocturnal hemoglobinuria

Parker

2016

Hematology

123

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Once suspected, the diagnosis of paroxysmal nocturnal hemoglobinuria (PNH) is straightforward when flow cytometric analysis of the peripheral blood reveals a population of glycosyl phosphatidylinositol anchor protein-deficient cells. But PNH is clinically heterogeneous, with some patients having a disease process characterized by florid intravascular, complement-mediated hemolysis, whereas in others, bone marrow failure dominates the clinical picture with modest or even no evidence of hemolysis observed. The clinical heterogeneity is due to the close, though incompletely understood, relationship between PNH and immune-mediated bone marrow failure, and that PNH is an acquired, nonmalignant clonal disease of the hematopoietic stem cells. Bone marrow failure complicates management of PNH because compromised erythropoiesis contributes, to a greater or lesser degree, to the anemia; in addition, the extent to which the mutant stem cell clone expands in an individual patient determines the magnitude of the hemolytic component of the disease. An understanding of the unique pathobiology of PNH in relationship both to complement physiology and immune-mediated bone marrow failure provides the basis for a systematic approach to management.

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“…To our knowl edge, this caseis the first reported use of a 7-French sheathedcurved metal cannula and flexible needle through a transhepatic ap proach for creating a hepatic venous outflow tract in a patient with Budd-Chiari syndrome. The curved 14-gauge cannula allowed the op eratorto steerthe needlein the coronalplane to a levelin theNC several centimeters below the right atrium.In thisway, myocardialper foration, previously reported as a complica tion ofhepatic venous stenting in Budd-Chiari syndrome [4], was avoided. Rotating the curved cannula also allowed the operator to steerthe needlein an axial plane, guiding it to ward the NC snare loop on orthogonal pro jections.The 5.2-Frenchcatheterandflexible needle combination allowed immediate pan sageofa guidewire into the NC oncethe nee dle was passedthrough the snare loop in the NC.…”

Section: Discussionmentioning

confidence: 99%

Percutaneous hepatic vein reconstruction for Budd-Chiari syndrome.

Suhocki

Trotter²

1998

American Journal of Roentgenology

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Short hepatic vein or inferior vena cava(IVC) occlusionsassociated with Budd-Chiari syndromeare usually treated by balloon dilatation and vas cular stent placement. Long-segment chronic occlusionscan make such revascularization techniques more challenging. We describe the transhepatic use of an angled transjugular liver biopsy cannula and a flexible tip needle for creating a tract between a right hepatic vein remnant and the IVC to restore hepatic vein outflow in a patient with Budd-Chiari syndrome. Rapid resolution of the patient's asciteswas associatedwith incarcerationof an umbilicalhernia. Subject and MethodsA 24-year-old woman with a 7-year histoty of pctlycythemiavera presentedwith abdominal disten

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Radiological intervention in budd-chiari syndrome: Techniques and outcome in 18 patients

Cited by 83 publications

References 26 publications

Diagnosis and management of paroxysmal nocturnal hemoglobinuria

Diagnosis and management of paroxysmal nocturnal hemoglobinuria

Update on the diagnosis and management of paroxysmal nocturnal hemoglobinuria

Percutaneous hepatic vein reconstruction for Budd-Chiari syndrome.

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