“…For example, the concentration of serum albumin is similar in cir- T.bil, serum total bilirubin; Plt, platelet; SD, standard deviation a Chest tube was not inserted in patient 1 rhotic patients with and without pleural effusion, and the other mechanisms cannot explain one-sided hydrothorax. At present, it is generally accepted that hepatic hydrothorax results from peritoneopleural communication, such that acsitic fluid directly flows into the pleural cavity via a diaphragmatic [1,2,4,15,16]. Although the clinical findings in some patients indicate acute accumulation of a large amount of pleural effusion soon after the reduction of ascites in cirrhotic patients, several investigators have reported the possible presence of a diaphragmatic defect in such patients, based on the following findings: (a) disappearance of the pleural effusion after the introduction of a pneumoperitoneum or positive pressure ventilation [4] and (b) scintigraphic diagnosis of the transdiaphragmatic movement of ascites after intraperitoneal radioisotope injection [2].…”