In recent years cardioneuroablation has emerged as a novel therapeutic
option for bradyarrhythmias, which can avoid necessity for pacemaker
implantation. Main assumption of cardioneuroablation is that destroying
epicardially located neural bodies of parasympathetic postganglionic
neurons by endocardial radiofrequency ablation would permanently
increase sinus rhythm. In some cases unipolar radiofrequency ablation
might damage only neural fibers and clinical effect of the procedure
might be transient due to subsequent reinnervation. Bipolar ablation can
create deeper, transmural and durable lesions. This property seems
promising as a method to overcome possible disadvantage of unipolar
cardioneuroablation. To our knowledge, we present first bipolar
cardioneuroablation.