QUANTIFICATION OF NEUROMETABOLITES IN SUBACUTE SCLEROSING PANENCEPHALITIS BY
            <sup>1</sup>
            H-MRS

Anlar, Banu

doi:10.1212/01.wnl.0000265409.38132.40

Neurology

2007

DOI: 10.1212/01.wnl.0000265409.38132.40

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QUANTIFICATION OF NEUROMETABOLITES IN SUBACUTE SCLEROSING PANENCEPHALITIS BY ¹ H-MRS

Banu Anlar

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Cited by 9 publications

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Blue Moon Neurovirology: The Merits of Studying Rare CNS Diseases of Viral Origin

O’Donnell

Rall

2010

J Neuroimmune Pharmacol

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While measles virus (MV) continues to have a significant impact on human health, causing 150,000-200,000 deaths worldwide each year, the number of fatalities that can be attributed to MV-triggered central nervous system (CNS) diseases are on the order of a few hundred individuals annually (World Health Organization 2009). Despite this modest impact, substantial effort has been expended to understand the basis of measles-triggered neuropathogenesis. What can be gained by studying such a rare condition? Simply stated, the wealth of studies in this field have revealed core principles that are relevant to multiple neurotropic pathogens, and that inform the broader field of viral pathogenesis. In recent years, the emergence of powerful in vitro systems, novel animal models, and reverse genetics has enabled insights into the basis of MV persistence, the complexity of MV interactions with neurons and the immune system, and the role of immune and CNS development in virus-triggered disease. In this review, we highlight some key advances, link relevant measles-based studies to the broader disciplines of neurovirology and viral pathogenesis, and propose future areas of study for the field of measles-mediated neurological disease.

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Blue Moon Neurovirology: The Merits of Studying Rare CNS Diseases of Viral Origin

O’Donnell

Rall

2010

J Neuroimmune Pharmacol

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Apoptosis in subacute sclerosing panencephalitis: Possibility for treatment

2013

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Evidence for neuroinflammation and neuroprotection in HCV infection-associated encephalopathy

Bokemeyer

Goldbecker

et al. 2010

Gut

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As the increase of choline, creatine and myo-inositol are usually interpreted to indicate glial activation and macrophage infiltration in chronic inflammation and slow virus infections of the brain the present data endorse the hypothesis, that HCV infection may induce neuroinflammation and brain dysfunction. The concomitant increase of NN and the negative correlation to the extent of fatigue suggest a cerebral compensatory process after HCV infection.

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QUANTIFICATION OF NEUROMETABOLITES IN SUBACUTE SCLEROSING PANENCEPHALITIS BY ¹ H-MRS

Cited by 9 publications

References 12 publications

Blue Moon Neurovirology: The Merits of Studying Rare CNS Diseases of Viral Origin

Blue Moon Neurovirology: The Merits of Studying Rare CNS Diseases of Viral Origin

Apoptosis in subacute sclerosing panencephalitis: Possibility for treatment

Evidence for neuroinflammation and neuroprotection in HCV infection-associated encephalopathy

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QUANTIFICATION OF NEUROMETABOLITES IN SUBACUTE SCLEROSING PANENCEPHALITIS BY 1 H-MRS

Cited by 9 publications

References 12 publications

Blue Moon Neurovirology: The Merits of Studying Rare CNS Diseases of Viral Origin

Blue Moon Neurovirology: The Merits of Studying Rare CNS Diseases of Viral Origin

Apoptosis in subacute sclerosing panencephalitis: Possibility for treatment

Evidence for neuroinflammation and neuroprotection in HCV infection-associated encephalopathy

Contact Info

Product

Resources

About

QUANTIFICATION OF NEUROMETABOLITES IN SUBACUTE SCLEROSING PANENCEPHALITIS BY ¹ H-MRS