Pyloric Stenosis in Pediatric Surgery

Pandya, Samir; Heiss, Kurt F.

doi:10.1016/j.suc.2012.03.006

Cited by 124 publications

(24 citation statements)

References 30 publications

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“…HPS has been linked to several genetic, environmental, and mechanical causative factors, [8][9][10][11][12][13] such as male predominance (4:1), twin birth, affected relatives, primigravida, cesarean section, born in summer or spring, formula feeding, and use of erythromycin. [6][7][8][9][10][11][12][13] Although not mentioned in the literature, we question if the number of general anesthetics or the type of EA repair could possibly be associated with each other.…”

Section: Introductionmentioning

confidence: 94%

“…[1][2][3][4][5] Hypertrophic pyloric stenosis (HPS) occurs in approximately one in 400 live births in the western population. 6,7 The pyloric muscle is hypertrophied and the pyloric channel becomes narrow and elongated, causing gastric outlet obstruction. Symptoms, such as vomiting, typically occur at 2 to 8 weeks of age and peak at 3 to 5 weeks.…”

Section: Introductionmentioning

confidence: 99%

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Increased Incidence of Hypertrophic Pyloric Stenosis in Esophageal Atresia Patients

et al. 2013

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Section: Introductionmentioning

confidence: 94%

Section: Introductionmentioning

confidence: 99%

Increased Incidence of Hypertrophic Pyloric Stenosis in Esophageal Atresia Patients

et al. 2013

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“…[128, 129] The 7.5% incidence of pyloric stenosis in EA patients reported by Van Beelan was 30 times higher than its 0.25% incidence in the normal population. [129] The diagnosis was generally delayed, by a median of 6 days (range, 1–21 days).…”

Section: Associated Gastrointestinal Conditions In Children With Eamentioning

confidence: 99%

How to Care for Patients with EA-TEF: The Known and the Unknown

Mousa

Krishnan

Hassan

et al. 2017

Curr Gastroenterol Rep

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EA patients are at high risk for gastroesophageal reflux and esophageal strictures, and the sequela that result. Extraintestinal manifestations of gastroesophageal reflux disease (GERD) can appear similar to other pathologic diagnoses commonly found in EA patients, such as congenital stricture, eosinophilic esophagitis, esophageal dysmotility, tracheomalacia, recurrent fistula, aspiration, etc. Therefore, it is important to have a standardized way to monitor for these issues. pH impedance allows for detection of nonacid reflux and the height of reflux, which are important in correlating symptoms with reflux episodes. A multidisciplinary approach is beneficial in evaluating and monitoring EA patients in the long term.

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“…Another condition occasionally reported in the FPIES caseloads is pyloric stenosis, suspected when symptoms of projectile and repetitive vomiting, eventually leading to dehydration and shock, occur in the first week of life [41]. Sometimes pyloric stenosis is associated, at physical examination, with the presence of a firm and nontender mass in the right upper quadrant, described as an ‘olive’.…”

Section: Acute and Chronic Food Protein-induced Enterocolitis Syndromementioning

confidence: 99%

Differential diagnosis of food protein-induced enterocolitis syndrome

Fiocchi

Claps

Dahdah

et al. 2014

Current Opinion in Allergy &Amp; Clinical Immunology

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Purpose of reviewTo assess all the possible differential diagnosis of food protein-induced enterocolitis syndrome (FPIES), both in acute and chronic presentation, reviewing the data reported in published studies.Recent findingsThere is an increase of reported cases of FPIES in recent years. As the disease presents with nonspecific symptoms, it can be misunderstood in many ways. The differential diagnosis includes, in acute presentations, the following: sepsis, other infectious diseases, acute gastrointestinal episodes, surgical emergencies, food allergies. In its chronic forms, FPIES may mimic malabsorption syndromes, metabolic disorders, primary immunodeficiencies, neurological conditions, coagulation defects, and other types of non-IgE-mediated food allergy.SummaryA thorough clinical evaluation, including symptoms, signs, and laboratory findings, is necessary to lead the clinicians toward the diagnosis of FPIES. The major reason for delayed diagnosis appears to be the lack of knowledge of the disease.

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Pyloric Stenosis in Pediatric Surgery

Cited by 124 publications

References 30 publications

Increased Incidence of Hypertrophic Pyloric Stenosis in Esophageal Atresia Patients

Increased Incidence of Hypertrophic Pyloric Stenosis in Esophageal Atresia Patients

How to Care for Patients with EA-TEF: The Known and the Unknown

Differential diagnosis of food protein-induced enterocolitis syndrome

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