Pulse pressure monitoring in hypertensive black Africans

Konin, C.; Adoh, M.; Adoubi, A.; Ekou, A.; Koffi, J.; Jj, N'djessan; A, Ahoua

doi:10.5830/cvja-2011-001

Cited by 1 publication

(2 citation statements)

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“…However, there have been scarcely documented isolated cases/series whereby underlying HIV was unfurled by CVT or known serologically positive HIV patients without co-existing opportunistic neuro-infection(s) who developed CVT. [3][4][5]17,18 Cryptococcal meningitisassociated CVT in HIV-infected patients has been a more common concern. 19 On occasions, in a few previous reports/series of HIV-infected individuals with CVT (without opportunistic neuro-infections), hyperhomocysteinemia, low B12 levels, chronic alcoholism, and abnormal levels of protein S have been recorded.…”

Section: Discussionmentioning

confidence: 99%

“…19 On occasions, in a few previous reports/series of HIV-infected individuals with CVT (without opportunistic neuro-infections), hyperhomocysteinemia, low B12 levels, chronic alcoholism, and abnormal levels of protein S have been recorded. 5,17,20 Now, whether these confounders are true contributing factors for the development of CVT or just mere epiphenomena remain elusive. 21…”

Section: Discussionmentioning

confidence: 99%

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Cerebral Venous Thrombosis as The Sole Presenting Manifestation of Human Immunodeficiency Virus and Hepatitis B Virus Co-Infection

Dutta

Ghosh

Pandit

et al. 2022

MRAJ

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Background: Cerebral venous thrombosis (CVT) following either human immunodeficiency virus (HIV) infection or hepatitis B virus (HBV) infection is a very rare condition. Moreover, it has never been reported as the presenting manifestation of HIV and HBV co-infection, even more so when the patient had a normal CD4 count and no demonstrable opportunistic infections. We aimed to report the first case of an adult Indian male, an intravenous drug abuser who developed CVT as the presenting manifestation of HIV-HBV co-infection. Methods: Patient data were obtained from medical records from the Bangur Institute of Neurosciences, Institute of Post Graduate Medical Education and Research & SSKM Hospital, Kolkata, West Bengal, India. Results: A 25-year-old male with a history of intravenous drug abuse and a normal CD4 count developed CVT as the presenting manifestation of HIV-HBV co-infection. His CD4 count was normal, and he had no demonstrable opportunistic infections. He had an uneventful recovery of the condition (CVT) following the institution of conventional anticoagulation therapy alongside anti-retroviral therapy. Conclusion: Whether illicit drug abuse or HIV/HBV infection itself or all in combination led to this thrombotic event cannot be precisely established. Notwithstanding, we recommend serologic testing for HIV and HBV in patients suffering from CVT with high-risk behavior.

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