Psychosocial Developments: Towards a Model of Recovery

Davis, Eric S.; Velleman, Richard; Smith, G. W.; Drage, Michael

doi:10.1002/9780470696101.ch1

Cited by 3 publications

(1 citation statement)

References 37 publications

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“…The schizophrenia concept and the criteria for diagnosis are built into diagnostic manuals and provide mental health nurses with a structure, consistency, familiarity and a model from which to deliver clinical interventions and research their role and interventions. Yet, acceptance of a genetic cause for schizophrenia is anathema to a person‐centred, evidenced‐based and recovery‐orientated mental health service (Davis et al . 2006).…”

Section: Introductionmentioning

confidence: 99%

Genes and schizophrenia: a pseudoscientific disenfranchisement of the individual

Fleming

Martin

2011

Journal of Psychiatric and Mental Health Nursing

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The biological model of schizophrenia remains the dominant model within mental health services. It has a powerful influence on the culture of mental health services; providing the structure for the delivery and selection of mental health treatments. There is widespread acceptance of a genetic cause for schizophrenia. Acceptance of a genetic cause is inconsistent with a person-centred recovery-orientated approach. The following paper provides a rigorous review of the underpinning research that supports the genetic argument. Appraisal of family, twin and adoption studies uncovers serious flaws in the methodologies and statistical analyses used in studies. These flaws not only artificially inflate the genetic contribution to schizophrenia but also invalidate many of the findings. More recent micro-imaging techniques have also failed to find replicable and consistent findings indicating a clear genetic pathway to schizophrenia. Freed from the implied pessimism of an unmodifiable genetic cause for schizophrenia, mental health nurses can confidently work to instil hope with people that have a diagnosis of schizophrenia.

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Section: Introductionmentioning

confidence: 99%

Genes and schizophrenia: a pseudoscientific disenfranchisement of the individual

Fleming

Martin

2011

Journal of Psychiatric and Mental Health Nursing

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Early Intervention and Vocational Opportunities

Davis

Burden

Manning

2010

Promoting Recovery in Early Psychosis

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From Classical Psychodynamics to Evidence Synthesis: The Motif of Repression and a Contemporary Understanding of a Key Mediatory Mechanism in Psychosis

Fleming

Martin

2012

Curr Psychiatry Rep

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The stress vulnerability model has proven to be a politically important model for two reasons. It has provided the framework that defines a temporal and dynamic process whereby a person's uniquely determined biopsychosocial vulnerability to schizophrenia symptoms interacts with his or her capacity to manage stress and the amount and type of stress experienced in such a way that the person experiences schizophrenia symptoms. Second, the development of this framework promoted the notion of inherited and acquired vulnerability. Implicit was that vulnerability was individually determined and that there was a role for psychosocial factors in the development/maintenance of schizophrenia symptoms. This proved to be a catalyst for the development of studies implicating psychosocial factors in the etiology of schizophrenia symptoms. Studies derived from cognitive-behavioral theories have proven the most successful in identifying thinking patterns, emotional disturbances, and neurocognitive and defensive vulnerability factors inherent in the development of schizophrenia symptoms. Historically, within the psychoanalytic school there has been debate regarding the role of repressive coping mechanisms in schizophrenia development. Psychoanalytic theories have always appeared incapable of providing etiologic explanations of schizophrenia symptoms, with the possible exception of Melanie Klein, than other more salient psychosocial schools. Mechanisms within the process of repressive coping are consistent with evidence and mechanisms supporting the stress vulnerability models and existing cognitive-behavioral theories regarding development of paranoid delusions. These mechanisms are less consistent with social cognitive explanations of schizophrenia symptoms.

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Psychosocial Developments: Towards a Model of Recovery

Cited by 3 publications

References 37 publications

Genes and schizophrenia: a pseudoscientific disenfranchisement of the individual

Genes and schizophrenia: a pseudoscientific disenfranchisement of the individual

Early Intervention and Vocational Opportunities

From Classical Psychodynamics to Evidence Synthesis: The Motif of Repression and a Contemporary Understanding of a Key Mediatory Mechanism in Psychosis

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