Progressing small vessel pontine infarction includes different etiologies

Nakase, Taizen; Sasaki, Masahiro; Ikeda, Yasuko; Suzuki, Akifumi

doi:10.1002/acn3.25

Cited by 13 publications

(6 citation statements)

References 18 publications

(32 reference statements)

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“…In addition, we also analyzed the correlation between different location of cerebral infarction and clinical progression of cerebral infarction in this study. The results showed that progressive cerebral infarction of perforating artery was mainly distributed in the areas of the para -body of lateral ventricle and pons, which was consistent with previous studies ( Li et al, 2020 , Nakase et al, 2014 ). The reason is that the supplying artery of the para -body of lateral ventricle is the superficial branch artery of the middle cerebral artery, such as the lenticulo-striate artery and the anterior choroid artery.…”

Section: Discussionsupporting

confidence: 92%

The value of diffusion weighted imaging in predicting the clinical progression of perforator artery cerebral infarction

Yang

Liu

et al. 2022

NeuroImage: Clinical

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Section: Discussionsupporting

confidence: 92%

The value of diffusion weighted imaging in predicting the clinical progression of perforator artery cerebral infarction

Yang

Liu

et al. 2022

NeuroImage: Clinical

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“…However, Multiple logistic regression results showed that the OR of BABD was much higher than the OR of infarct volume in our study,So We speculated that subtype-BABD has a greater impact on the occurrence of END. Interestingly, another study [ 8 ] suggested that END was not related to the size of either infarct; however, its sample size was relatively small ( n = 38), the expansion of ischemic lesions was not correlated with END, and the actual lesion size was not measured.…”

Section: Discussionmentioning

confidence: 99%

“…In previous studies [ 3 , 7 ], END was reported to be related to the topographic location of the pontine infarction. However, another study showed that END was independent of the location and was not correlated with the size of the infarct [ 8 ]. Recently, the infarct size rather than topographic location of pontine infarction was suggested to be a possible predictor of END [ 9 ].…”

Section: Introductionmentioning

confidence: 99%

Risk factors for early neurological deterioration in acute isolated pontine infarction without any causative artery stenosis

Liu

Peng²,

Wang

et al. 2022

BMC Neurol

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Background This study aimed to investigate the risk predictors for early neurological deterioration (END) in isolated acute pontine infarction without any causative artery stenosis. Methods In this retrospective study, patients with isolated acute pontine infarction within 72 h of symptom onset were enrolled between October 2017 and December 2021. END was defined as an increase in the National Institutes of Health Stroke Scale (NIHSS) score ≥ 2 points within the first week postadmission. Patients were divided into the END and the non-END groups. Multiple logistic regression analysis was used to evaluate independent predictors of END in patients with isolated acute pontine infarction. Results A total of 153 patients were included in the final study (62 females; mean age, 67.27 ± 11.35 years), of whom 28.7% (47 of 153) experienced END. Multiple logistic regression analyses showed that infarct volume (adjusted odds ratio [aOR], 1.003; 95% CI, 1.001–1.005; P = 0.002) and basilar artery branch disease (aOR, 3.388; 95% CI, 1.102–10.417; P = 0.033) were associated with END. The combined ROC analysis of the infarct volume and basilar artery branch disease for predicting END showed that the sensitivity and specificity were 80.9% and 72.6%, respectively. Conclusion Basilar artery branch disease and infarct volume were associated with END in acute isolated pontine infarction and may be useful prognostic factors for neurological progression.

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“…Tegmental infarction usually represents SAD, and most unisolated pontine infarctions were caused by SAD in our study. Bilateral pontine infarction indicates severe neurological dysfunction and is related to poor outcomes [17, 18]. Claudio et al showed that 11% of isolated pontine infarctions were bilateral ventral infarctions [8].…”

Section: Discussionmentioning

confidence: 99%

Topographic location of unisolated pontine infarction

et al. 2019

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Background The topographic location of acute pontine infarction is associated with clinical syndromes and prognosis. Previous studies focused on isolated pontine infarction, but the topographic location of unisolated pontine infarction has remained unclear. Methods This was a prospective, multicenter, longitudinal registry study. Patients with acute pontine infarction confirmed by magnetic resonance imaging (MRI) were enrolled. Based on the territory of the pontine artery, the topographic location was divided into anteromedial, anterolateral, tegmental, bilateral and unilateral multiple infarctions. Results From May 1, 2003, to Oct 31, 2017, 1003 patients were enrolled, and 330 had unisolated pontine infarction. For isolated pontine infarction, 44.9, 19.8, 16.0, 13.1 and 6.2% of patients had anteromedial, anterolateral, tegmental, bilateral and unilateral multiple pontine infarctions, respectively. For unisolated pontine infarction, 30.3, 19.7, 24.5, 15.2 and 10.3% of patients had anteromedial, anterolateral, tegmental, bilateral and unilateral multiple pontine infarctions, respectively. Conclusion In this large series study, our data revealed fewer anteromedial infarctions and more tegmental and unilateral multiple infarctions in patients with unisolated pontine infarction than in patients with isolated pontine infarction.

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Progressing small vessel pontine infarction includes different etiologies

Cited by 13 publications

References 18 publications

The value of diffusion weighted imaging in predicting the clinical progression of perforator artery cerebral infarction

The value of diffusion weighted imaging in predicting the clinical progression of perforator artery cerebral infarction

Risk factors for early neurological deterioration in acute isolated pontine infarction without any causative artery stenosis

Topographic location of unisolated pontine infarction

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