Prognosis of ramsay hunt syndrome presenting as cranial polyneuropathy

Abstract

The tracheal luminal contour and functional epithelial regeneration without graft rejection and inflammation were observed after repair of a tracheal resection using allogeneic implants with chondrocytes cultured with fibrin/HA.

A case of herpes zoster oticus with multiple cranial nerve palsy

“…The occurrence rate of associated cranial polyneuropathy has been reported to be 1.8-3.2% and cranial nerves VII, VIII, IX and X are the ones most commonly affected. 6,7 In present case, the skin lesions which were confined to left pinna with associated LMN type of left facial palsy was the key to the diagnosis of RHS. Besides these abnormalities, oropharyngeal discomfort and complaints such as dysphagia and hoarseness were more troublesome symptom due to paralysis of the left sided pharyngeal muscles.…”

A rare case of herpes zoster maxillaris with cranial polyneuropathy

“…The occurrence rate of associated cranial polyneuropathy has been reported to be 1.8-3.2% and cranial nerves VII, VIII, IX and X are the ones most commonly affected. [10][11][12] In our case, the skin lesions were absent, but there was mucosal lesions confined to left side of the palate with associated LMN type of left sided facial palsy and involvement of ipsilateral IX, X and XII cranial nerves. Besides these, oropharyngeal discomfort and complaints such as dysphasia and pain inside mouth were more troublesome symptom due to paralysis of the left sided pharyngeal muscles and the palatal lesions.…”

Cranial Polyneuropathy in Ramsay Hunt Syndrome Manifesting Severe Pharyngeal Dysphagia: a Case Report and Literature Review

“…Concurrent involvement of CNs such as CN V, CN IX, CN X, CN XI, and CN XII is rare, but leads to various symptoms of neurological deficits including impaired eye movement, trigeminal neuralgia, swallowing difficulty, hoarseness, and tongue deviation. Although the mechanisms are unclear, vasculitis in the carotid, middle meningeal, and ascending pharyngeal arteries, which can be infected by the direct perineural spread of the VZV along anastomotic pathways, may contribute to the development of cranial polyneuropathy in RHS [4]. The involvement of multiple CNs in RHS results in greater loss of function and is associated with a poor prognosis relative to facial nerve palsy regardless of the initial HBS grade.…”