1936
DOI: 10.1177/003591573602900418
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Problems of Obsessional Illness

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Cited by 193 publications
(71 citation statements)
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“…Most of these associations have been reported for both adult and adolescent patients but adolescent cases seem particularly wellrepresented in most series. Aubrey Lewis (1936) for example, noted several cases with childhood onset following encephalitis or febrile seizures. Obsessive phenomena have long been recognised in association with seizures, particuarly temporal lohe epilepsy (Bear & Fedio, 1977).…”
Section: Association With Other Conditionsmentioning
confidence: 99%
“…Most of these associations have been reported for both adult and adolescent patients but adolescent cases seem particularly wellrepresented in most series. Aubrey Lewis (1936) for example, noted several cases with childhood onset following encephalitis or febrile seizures. Obsessive phenomena have long been recognised in association with seizures, particuarly temporal lohe epilepsy (Bear & Fedio, 1977).…”
Section: Association With Other Conditionsmentioning
confidence: 99%
“…For example, Lewis (1936) claimed that "the recognition of the obsession as senseless is not an essential characteristic" and recognition that it is absurd.is not always present" (cited in Catapano, Sperandeo, Perris, Lanzaro, & Maj, 2001). This observation was corroborated by the field trial of Foa and Kozak (1995), which documented that there are OCD patients who lack insight into their symptoms, and suggested that insight in OCD lies on a continuum ranging from good to poor.…”
Section: Introductionmentioning
confidence: 97%
“…Psychiatry, 1990 ,29,3:407-412 . Key Wo rd s : ge netic , familia l, obsessi ve compul sive disorde r (OCD) .Since the classic report of Au brey Lew is in 1936 , seve ra l cli nica l investiga tors have reported increas ed prevalence of obsess ive co mpulsive disorder (OC D) among first-d egree relatives of obsessive co mpulsive (OC) pati ent s , (0 % to 40 %) (Lewis , 1936;Brown , 1942; Rud in , 1953; Rasmussen and Ts uang , 1986) , although others have found no increases (Ros enberg, 1967; Coppen et aI., 1965; McK eon and Murray , 1987). Rates higher than expected in relatives sugges t a ge netic co ntribution to etio logy and furth er suppor t is provided by repor ts of grea ter co ncorda nce for OCD among monozygot ic twin s than dizygot ic twins (Ino uye, 1965; McKeon ct al.…”
mentioning
confidence: 99%