Primary osteoarthritis of the lumbar spine presenting as chronic low back pain or sciatica or both (prolapsed lumber disc syndrome): establishing its cause, pathogenesis, and treatment: A randomized case-control study

Abstract

: The objectives of this study were to compare three scores, Western Ontario McMaster Universities Osteoarthritis Index (WOMAC), Oswestry Disability Index (ODI) for low back pain, and Visual Analogue Scale (VAS) Vertical Version at the beginning and end point in two, Trial (G1), and Control (G2) groups of Primary Osteoarthritis of lumbar spine patients.The inner nucleus pulposus may rupture out from the annulus as the disc continues to degrade or as the spine continues to be stressed. This is a herniated or burst disc. The nerve roots that are immediately behind the disc space may then be pressed by the disc material fragments. This may result in discomfort, frailty, numbness, or alterations in feeling.: Understanding the causes, pathophysiology, and treatments for prolapsed lumber disc syndrome, which presents as primary osteoarthritis of the lumbar spine and causes chronic low back pain, sciatica, or both.: In this study, the total number of patients were 150, in G1 - 100, and G2 - 50. G1 received the hypothesized treatment, Contracture Correction Therapy (CCT), while G2 did “No Therapy”. WOMAC determination was done by the questionnaire; ODI by modified Oswestry Low Back pain disability questionnaire and VAS by Visual Analogue Scale Vertical Version, all at 0, 6, 12, and 24 weeks. The CCT consisted of 1. Immobilization of the spine. 2. Passive and active extension of the spine. 3. Three therapeutic rituals and 4. Advice to avoid stooping.: The CCT receiving was associated with much recovery (P <0.00) while non-receiving with much less (P =0.00). In G1 WOMAC decreased from 54.29 to 0.00; ODI from 51.82 to 1.80; VAS rose from 37.56 to 100 (rise indicates betterment) P= 0.00. In G2 WOMAC lowered from 56.40 to 4.08; ODI from 65.10 to 6.0; VAS rose from 34.40 to 81.00. Relief in symptoms was similar.: The cause, pathogenesis, and treatment are deficient full extension, I.V. joint capsule contracture formation, and extension of the lumbar spine respectively.