Primary aortoenteric fistula: a rare complication
of an eroding duodenal stent

Kehagias, Dimitrios; Mulita, Francesk; Marlafeka, Ioanna; Verras, Georgios-Ioannis; Kehagias, Ioannis

doi:10.5114/kitp.2022.119768

Cited by 1 publication

(1 citation statement)

References 6 publications

(8 reference statements)

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“…This integrated approach aims to prevent aneurysm enlargement, reduce the risk of rupture, and intervene prior to the development of dissection or rupture. In addition to the common complications of rupture and bleeding, TAA can also lead to uncommon intestinal fistulas, where aortic inflammation and direct friction collaborate to spontaneously form primary fistulas [ 28 , 29 ]. The infiltration of immune cells could potentially explain the structural changes in blood vessel walls.…”

Section: Discussionmentioning

confidence: 99%

The causal relationship between thoracic aortic aneurysm and immune cells: a mendelian randomization study

Liu,

Pan,

Xia

et al. 2024

BMC Cardiovasc Disord

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One of the pathogenic causes of thoracic aortic aneurysm (TAA), a dangerous vascular condition that can cause aortic rupture, is autoimmune disorders. Currently, immune cell clustering is becoming more and more refined, and the specific immune cell phenotypes involved are yet unknown. Here, we want to clarify the causal link between TAA risk and 731 immune cell traits. There was a Mendelian randomization analysis (MR). We discovered that the presence of TAA led to an increase in CD45 on CD33− HLA-DR− myeloid cells, an increase in CD45 on natural killer cells, and a decrease in FSC-A on granulocytes after applying FDR correction. Our research also revealed a strong correlation between the incidence of TAA and an increase in immune cells with CD3 on CD39+ CD4+, and CD25 on IgD− CD27− phenotypes. Through genetic techniques, our research has shown the intimate relationship between immune cells and TAA, offering direction for future clinical investigations.

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Section: Discussionmentioning

confidence: 99%