Prevalence of food allergy in New York City school children

Taylor-Black, Sarah A.; Mehta, Harshna; Weiderpass, Elisabete; Boffetta, Paolo; Sicherer, Scott H.; Wang, Julie

doi:10.1016/j.anai.2014.03.020

Cited by 16 publications

(4 citation statements)

References 10 publications

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“…In New York City, physician-diagnosed FA was significantly higher (17.5%) among private school respondents compared with public school respondents (7.4%) 98 5. The prevalence of physician-diagnosed FA in urban minority children was 3.4%, significantly lower than reported national estimates 99 1. Disparities in FA management have been documented, with black and Hispanic parents less likely to correctly identify signs of FA reactions and less likely to identify food triggers 101 2.…”

Section: Disparities In Fa What Is Known?contrasting

confidence: 59%

“…A cross-sectional study compared 2 private and 2 public charter schools in New York City, NY, that differed in racial/ ethnic composition and SES, with more black and lower income children in public charter schools. 99 Physician-diagnosed FA was significantly higher (17.5%) among private school respondents compared with public school respondents (7.4%). Even among those with a history of severe reaction to food, nearly half the children in the public schools lacked a physician diagnosis.…”

Section: Disparities In Fa What Is Known?mentioning

confidence: 85%

See 1 more Smart Citation

Health disparities in allergic and immunologic conditions in racial and ethnic underserved populations: A Work Group Report of the AAAAI Committee on the Underserved

Davis

Apter

Casillas³

et al. 2021

Journal of Allergy and Clinical Immunology

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Section: Disparities In Fa What Is Known?contrasting

confidence: 59%

Section: Disparities In Fa What Is Known?mentioning

confidence: 85%

Health disparities in allergic and immunologic conditions in racial and ethnic underserved populations: A Work Group Report of the AAAAI Committee on the Underserved

Davis

Apter

Casillas³

et al. 2021

Journal of Allergy and Clinical Immunology

Self Cite

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“…Fox et al 45 noted in a UK allergy clinic from 1990-2004 an increase in the proportion of nonwhite patients with peanut allergy (but not egg allergy) from 26.8% to 50.3%, but the proportion of black subjects attending the clinic had not changed. Taylor-Black et al 46 investigated food allergy in New York City schools and did not identify a difference in food allergy rates between black and white children.…”

Section: Epidemiology and Natural History Prevalencementioning

confidence: 99%

Food allergy: A review and update on epidemiology, pathogenesis, diagnosis, prevention, and management

Sicherer

Sampson

2018

Journal of Allergy and Clinical Immunology

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1,216

977

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This review provides general information to serve as a primer for those embarking on understanding food allergy and also details advances and updates in epidemiology, pathogenesis, diagnosis, and treatment that have occurred over the 4 years since our last comprehensive review. Although firm prevalence data are lacking, there is a strong impression that food allergy has increased, and rates as high as approximately 10% have been documented. Genetic, epigenetic, and environmental risk factors are being elucidated increasingly, creating potential for improved prevention and treatment strategies targeted to those at risk. Insights on pathophysiology reveal a complex interplay of the epithelial barrier, mucosal and systemic immune response, route of exposure, and microbiome among other influences resulting in allergy or tolerance. The diagnosis of food allergy is largely reliant on medical history, tests for sensitization, and oral food challenges, but emerging use of component-resolved diagnostics is improving diagnostic accuracy. Additional novel diagnostics, such as basophil activation tests, determination of epitope binding, DNA methylation signatures, and bioinformatics approaches, will further change the landscape. A number of prevention strategies are under investigation, but early introduction of peanut has been advised as a public health measure based on existing data. Management remains largely based on allergen avoidance, but a panoply of promising treatment strategies are in phase 2 and 3 studies, providing immense hope that better treatment will be imminently and widely available, whereas numerous additional promising treatments are in the preclinical and clinical pipeline.

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“…In a study that included 2 public schools and 2 charter schools from New York, Taylor-Black et al did not find a higher rate of FA among Black children compared to White children. 130 A recent meta-analysis by Greenhawt et al concluded that though there was some evidence of increased rates of food sensitization and FA among the Black population, the available data were still insufficient to label Black race as a risk factor for FA. 131 …”

Section: Gene-environment Interactions In Atopic Disordersmentioning

confidence: 99%

Resolving the etiology of atopic disorders by using genetic analysis of racial ancestry

Gupta

Johansson

Bernstein

et al. 2016

Journal of Allergy and Clinical Immunology

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Atopic dermatitis (AD), food allergy (FA), allergic rhinitis (AR) and asthma are common atopic disorders of complex etiology. The frequently observed “atopic march” from early AD to asthma and/or AR later in life as well as the extensive comorbidity of atopic disorders, suggests common causal mechanisms in addition to distinct ones. Indeed, both disease-specific and shared genomic regions exist for atopic disorders. Their prevalence also varies among races; for example, AD and asthma have a higher prevalence in African-Americans when compared to European-Americans. Whether this disparity stems from true genetic or race-specific environmental risk factors or both is unknown. Thus far, the majority of the genetic studies on atopic diseases have utilized populations of European ancestry, limiting their generalizability. Large cohort initiatives and new analytic methods such as admixture mapping are currently being employed to address this knowledge gap. Here we discuss the unique and shared genetic risk factors for atopic disorders in the context of ancestry variations, and the promise of high-throughput “-omics” based systems biology approach in providing greater insight to deconstruct into their genetic and non-genetic etiologies. Future research will also focus on deep phenotyping and genotyping of diverse racial ancestry, gene-environment, and gene-gene interactions.

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Prevalence of food allergy in New York City school children

Cited by 16 publications

References 10 publications

Health disparities in allergic and immunologic conditions in racial and ethnic underserved populations: A Work Group Report of the AAAAI Committee on the Underserved

Health disparities in allergic and immunologic conditions in racial and ethnic underserved populations: A Work Group Report of the AAAAI Committee on the Underserved

Food allergy: A review and update on epidemiology, pathogenesis, diagnosis, prevention, and management

Resolving the etiology of atopic disorders by using genetic analysis of racial ancestry

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