Prevalence and risk factors of work related asthma by industry among United States workers: data from the third national health and nutrition examination survey (1988–94)

Arif, Ahmed A.; Whitehead, Lawrence W.; Delclos, George L.; Tortolero, Susan R.; Lee, E S

doi:10.1136/oem.59.8.505

Cited by 75 publications

(69 citation statements)

References 41 publications

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“…This conspicuous difference has raised several important questions [28,65,66,69]. A possible explanation for the higher estimates of risk of asthma attributable to work exposure based on data derived from community-based studies is that such studies are more likely to include all individuals ever exposed in at-risk workplaces, rather than those currently exposed, thus reducing survivor effects.…”

Section: Asthma Attributable To Work Exposuresmentioning

confidence: 99%

“…There is insufficient evidence about causation of asthma from long-term "low-dose" exposure to irritants. Several recent studies have found an increased risk of asthma in jobs associated with irritant exposures, in particular among construction and textile workers [70], cleaners [27,54], shoemakers, metalplating workers and electrical machinery workers [28]. An increased risk of asthma in professions not normally associated with OA has led to the suspicion that proteins derived from ubiquitous agents may be responsible.…”

Section: Asthma Attributable To Work Exposuresmentioning

confidence: 99%

“…These surveys have been restricted to individual workplaces or to groups of individuals from the same occupation [24]. More recently, population-based studies have been undertaken to estimate the prevalence of OA [25][26][27]; in addition, data from population surveys have been analysed to derive such estimates [28]. These results are helpful in providing information about the size of the problem in high-risk workplaces, although they are potentially prone to survivor bias.…”

Section: Prevalence Of Occupational Asthmamentioning

confidence: 99%

See 2 more Smart Citations

Controversies in epidemiology of occupational asthma

Gautrin¹,

Aj²,

Nordman³

et al. 2003

Eur Respir J

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high-risk workplaces, medicolegal statistics and sentinel programmes indicate that y10% of adult-onset asthma is attributable to the workplace. The strategy to identify cases through questionnaires and tools that address functional, immunological and physiopathological issues needs to be improved.Although few in number and limited to a handful of workplaces, cohort studies found that the risk of developing occupational asthma is determined less by individual susceptibility (e.g. atopy, tobacco smoking, human leukocyte antigen phenotype) and more by the level of exposure to its causes; in general, the higher the exposure, the greater the risk, and, by implication, lowering the level of exposure reduces the incidence of disease.Occupational asthma can be used as a satisfactory model for the development of adult-onset asthma. There is a great need to develop intervention strategies through adequate surveillance programmes in high-risk workplaces. Eur Respir J 2003; 22: 551-559. Epidemiology of occupational asthma: general considerations on various study designs and methodological approachesEpidemiology is the study of the distribution, determinants and outcome of disease. In this article, the authors will describe recently acquired knowledge of the epidemiology of occupational asthma, as well as current areas of controversy.BECKLAKE et al.[1] have discussed the features, strengths and weaknesses of several designs used in epidemiological studies. These designs include the randomised control trial, the prospective cohort, the case-referent study, the crosssectional (prevalence) study and the case series. Which design to use in any particular situation is a matter of judgment, determined by a number of factors that may include resources, cost, collaboration of employers and employees, and the question under study. So far, primarily for practical convenience, most epidemiological studies of occupational asthma have been cross-sectional in type. As discussed in several reviews [1,2], this type of study, primarily in the case of occupational asthma (OA), suffers from survivor bias, whose magnitude can be difficult to quantify. Randomised control trials are usually not feasible and are probably unethical in workplace studies, but quasi-experimental studies can provide results of high internal and external validity. Prospective cohort studies are more expensive than cross-sectional studies but are more powerful and less likely to be affected by selection or survivor biases; in particular, it is often possible to track the participants who leave the cohort and determine the possible effect of attrition on the estimates of both the frequency of OA and exposure/effect relationships [3]. This type of study has recently been successfully applied in cohorts of young apprentices [4][5][6][7][8][9][10]. Some case-referent studies have been carried out in the field of OA [11,12]. These studies are valuable in identifying and quantifying risk factors. Finally, there are many case series that mainly describe clinical cases (often si...

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Section: Asthma Attributable To Work Exposuresmentioning

confidence: 99%

Section: Asthma Attributable To Work Exposuresmentioning

confidence: 99%

Section: Prevalence Of Occupational Asthmamentioning

confidence: 99%

See 1 more Smart Citation

Controversies in epidemiology of occupational asthma

Gautrin¹,

Aj²,

Nordman³

et al. 2003

Eur Respir J

View full text Add to dashboard Cite

show abstract

“…This could partly be due to the healthy worker effect. An increased prevalence of respiratory symptoms was found in textile, workers in a textile industry are potentially exposed to cotton and other fiber dusts, textile dyes, and fabric printing and treatment chemicals 24) .…”

Section: Prevalence Of Respiratory Symptoms Industries In Iran 221mentioning

confidence: 99%

Prevalence of Respiratory Symptoms among Workers in Industries of South Tehran, Iran

Ghasemkhani

Kumashiro²,

Rezaei

et al. 2006

Ind Health

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“…There is now substantial evidence of an increased risk of asthma in domestic and office cleaning workers that is likely to represent a direct causal effect of chemical exposure associated with these occupations [3]. Additionally, other observational and case-control studies have reported positive associations between workplace exposure and a range of outcomes including asthma [4,5], occupational asthma [6][7][8], airway inflammation [9] and chronic obstructive pulmonary disease (COPD) [10]. A population-based case-control study in Australia [11] has recently reported higher levels of domestic exposure to volatile organic chemicals (VOCs) in children with asthma compared with controls.…”

mentioning

confidence: 99%

Household chemicals, persistent wheezing and lung function: effect modification by atopy?

Henderson¹,

Sherriff²,

Farrow³

et al. 2008

European Respiratory Journal

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The aims of the present study were to assess the effects of maternal use of domestic chemicals during pregnancy on wheezing and lung function in children aged f8.5 yrs and to explore the potential modifying effect of atopy.In the Avon Longitudinal Study of Parents and Children, a cohort study, a maternal composite household chemical exposure (CHCE) score was derived. Wheezing phenotypes from birth to age 7 yrs were assigned on the basis of reported wheeze. Lung function (forced expiratory volume in one second (FEV1), forced vital capacity (FVC), forced midexpiratory flow between 25 and 75% of FVC (FEF25-75%)) was measured at age 8.5 yrs; and atopy by skin-prick tests at age 7.5 yrs. Multinomial logistic and linear regression models assessed the relationship between wheezing outcomes, lung function and CHCE score, and interactions with atopy.Increased CHCE score was associated with early-(,18 months) and intermediate-(18-30 months) persistent and late-onset (.30 months) wheezing in nonatopic children (adjusted odds ratio per z-score of CHCE (95% confidence interval) 1.41 (1.13-1.76), 1.43 (1.02-2.13) and 1.69 (1.19-2.41), respectively). Increasing CHCE score was associated with decrements in FEV1 and FEF25-75%.Higher domestic chemical exposure during pregnancy was associated with persistent wheeze and lung function abnormalities in nonatopic children. This may result from pre-natal developmental effects or post-natal irritant effects on the developing airway, but is unlikely to be mediated through increased hygiene in the home.

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Prevalence and risk factors of work related asthma by industry among United States workers: data from the third national health and nutrition examination survey (1988–94)

Cited by 75 publications

References 41 publications

Controversies in epidemiology of occupational asthma

Controversies in epidemiology of occupational asthma

Prevalence of Respiratory Symptoms among Workers in Industries of South Tehran, Iran

Household chemicals, persistent wheezing and lung function: effect modification by atopy?

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