PREPARED: PREParation for Angiography in REnal Dysfunction

Taylor, Allen J.; Hotchkiss, David R.; Morse, R. W.; McCabe, John C.

doi:10.1378/chest.114.6.1570

Cited by 192 publications

(38 citation statements)

References 11 publications

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“…The first study (n = 36), published in 1998, reported that oral hydration was equally effective as IV hydration [15]. However, in this study, postprocedural IV hydration was also applied in the oral hydration group.…”

Section: Discussionmentioning

confidence: 97%

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Efficacy of Oral Hydration in the Prevention of Contrast-Induced Acute Kidney Injury in Patients Undergoing Coronary Angiography or Intervention

Akyüz¹,

Karaca²,

Öz³

et al. 2014

Nephron Clin Pract

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Background: Efficacy of intravenous (IV) volume expansion in preventing contrast-induced acute kidney injury (CI-AKI) is well known. However, the role of oral hydration has not been well established. The aim of this work was to evaluate the efficacy of oral hydration in preventing CI-AKI. Methods: We prospectively randomized 225 patients undergoing coronary angiography and/or percutaneous coronary intervention in either oral hydration or IV hydration groups. Patients who have at least one of the high-risk factors for developing CI-AKI (advanced age, type 2 diabetes mellitus, anemia, hyperuricemia, a history of cardiac failure or systolic dysfunction) were included in the study. All patients had normal renal function or stage 1-2 chronic kidney disease. Patients in the oral hydration group were encouraged to drink unrestricted amounts of fluids freely whereas isotonic saline infusion was performed by the standard protocol in the IV hydration group. Results: CI-AKI occurred in 8/116 patients (6.9%) in the oral hydration group and 8/109 patients (7.3%) in the IV hydration group (p = 0.89). There was also no statistically significant difference between the two groups when different CI-AKI definitions were taken into account. Conclusion: Oral hydration is as effective as IV hydration in preventing CI-AKI in patients with normal kidney function or stage 1-2 chronic kidney disease, and who also have at least one of the other high-risk factors for developing CI-AKI.

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Section: Discussionmentioning

confidence: 97%

“…However, four of these eight studies had more strength than the present study with respect to inclusion of patients with an eGFR <60 ml/min/1.73 m 2 [15,17,19,20]. …”

Section: Discussionmentioning

confidence: 99%

Efficacy of Oral Hydration in the Prevention of Contrast-Induced Acute Kidney Injury in Patients Undergoing Coronary Angiography or Intervention

Akyüz¹,

Karaca²,

Öz³

et al. 2014

Nephron Clin Pract

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“…Since not all of the intravenously administered isotonic crystalloid remains in the vascular space, in order to achieve a urine flow rate of at least 150 ml/h, ≥1.0–1.5 ml/kg/min of intravenous fluid has to be administered for 3–12 h before and 6–12 h after contrast exposure. Oral volume expansion may have some benefit, but there is not enough evidence to show that it is as effective as intravenous volume expansion [62]. …”

Section: Volume Expansionmentioning

confidence: 99%

Radiocontrast-Induced Acute Kidney Injury

McCullough¹

2008

Nephron Physiol

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Many radiographic studies and procedures use iodinated contrast media and consequently pose the risk of contrast-induced acute kidney injury (AKI). This is an important complication, which accounts for a significant number of cases of hospital-acquired renal failure associated increased hospital length of stay and increased mortality. Sustained reductions in renal blood flow, hypoxic injury, direct cellular toxicity by the contrast media, and superimposed organ injury are all believed to play a role in this form of AKI. Avoidance of dehydration and multimodality prevention measures may reduce rates of this problem in patients at risk. Contrast-induced AKI is likely to remain a significant challenge for specialists in the future since the patient population is aging, chronic kidney disease and diabetes are coming more common, and use of iodinated contrast is growing.

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“…But a definition of vivax-induced AKI is still unknown to us. In the RIFLE (Risk, Injury, Failure, Loss, End-stage kidney disease) classification of AKI, the risk stage is defined as an increase of serum creatinine with of 50% corresponding to a decrease in GFR, relative to baseline, of >25% or a urine output of <0.5 ml/kg/h for >6 h. Recently, the definition of risk was expanded to include an absolute increase in serum creatinine of 0.3 mg/dl or more [9, 10], and both glomerular and tubular dysfunction were suggested if the proteins and albumin in urine were elevated [11, 12]. In this study, nephropathy was classified by the degree of kidney damage (as measured by the level of proteinuria) and an absolute increase in serum creatinine of 0.3 mg/dl or more (equal to an eGFR <80 ml/min).…”

Section: Discussionmentioning

confidence: 99%

Predictors of <i>Plasmodium vivax</i> Malaria-Induced Nephropathy in Young Korean Men

Chung

Lee²,

Lee³

et al. 2008

Nephron Clin Pract

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Background:Plasmodium vivax malaria accounts for more than half of all malaria cases in Asia and Latin America. Despite the high prevalence of disease caused by this parasite, research into its effects (especially its renal effect) has lagged disproportionately. To investigate predictors of vivax malaria-induced nephropathy, we analyzed the cases of vivax malaria-induced nephropathy in young Korean men. Methods: This was a retrospective analysis of P. vivax patients with acute nephropathy (all males, n = 75), defined by an absolute increase in serum creatinine of 0.3 mg/dl or more (equal to an estimated glomerular filtration rate (eGFR) <80 ml/min) (group 1, n = 31) or proteinuria (group 2, n = 44), between January 2004 and December 2007. The eGFR was calculated using a simplified Modification of Renal Disease (MDRD) equation. None of the patients had a history of traveling abroad, drug abuse or blood transfusion. The clinical manifestations, laboratory abnormalities and predictors of nephropathy were reviewed. Results: Out of 398 cases of vivax malaria, 75 patients (all males) suffered from to vivax malaria-induced acute nephropathy. The mean age of the patients who were divided into two groups was 22.8 ± 3.7 and 21.6 ± 1.8 years, respectively (p = 0.089). In group 1, the total serum bilirubin significantly correlated with serum creatinine and eGFR (p = 0.004 and 0.035, correlation coefficient = 0.508 and –0.387, respectively). In group 2, 24-hour proteinuria significantly correlated with hemoglobin (p = 0.004, correlation coefficient = –0.424). Conclusion: Total serum bilirubin (group 1, an absolute increase in serum creatinine of 0.3 mg/dl or more (equal to an eGFR <80 ml/min)) and hemoglobin (group 2, proteinuria) are useful to predict vivax-induced nephropathy.

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PREPARED: PREParation for Angiography in REnal Dysfunction

Cited by 192 publications

References 11 publications

Efficacy of Oral Hydration in the Prevention of Contrast-Induced Acute Kidney Injury in Patients Undergoing Coronary Angiography or Intervention

Efficacy of Oral Hydration in the Prevention of Contrast-Induced Acute Kidney Injury in Patients Undergoing Coronary Angiography or Intervention

Radiocontrast-Induced Acute Kidney Injury

Predictors of <i>Plasmodium vivax</i> Malaria-Induced Nephropathy in Young Korean Men

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