Predictors of Outcomes of Contrast-Induced Acute Kidney Injury After Percutaneous Coronary Intervention in Patients With Chronic Kidney Disease

Kim, Ji‐Hwan; Yang, Jeong Hoon; Choi, Seung–Hyuk; Song, Young Bin; Hahn, Joo‐Yong; Choi, Jin-Ho; Lee, Sang Hoon; Gwon, Hyeon‐Cheol

doi:10.1016/j.amjcard.2014.09.022

Cited by 43 publications

(39 citation statements)

References 23 publications

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“…Indeed, previous studies had already reported the association between female sex and CI-AKI. [18][19][20][21][22] In their study focused on liver transplanted patients, Hilmi et al [23] stressed that all the women enrolled were in menopausal age and thus had lost the beneficial protective effects exerted by estrogens against cardiovascular and renal diseases, and would therefore be more exposed to various kidney insults. Of note and in accordance with this hypothesis, all female patients who developed CI-AKI in our study were in menopause.…”

Section: Discussionmentioning

confidence: 99%

Acute kidney injury in cirrhotic patients undergoing contrast-enhanced computed tomography

Filomia¹,

Maimone²,

Saitta³

et al. 2015

Digestive and Liver Disease

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Section: Discussionmentioning

confidence: 99%

Acute kidney injury in cirrhotic patients undergoing contrast-enhanced computed tomography

Filomia¹,

Maimone²,

Saitta³

et al. 2015

Digestive and Liver Disease

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“…Short and long term mortalities of patients with CI-AKI have been shown to be higher compared with patients without CI-AKI[85-87]. However there are few studies about the long-term renal prognosis of patients who developed CI-AKI.…”

Section: Prognosis Of Ci-akimentioning

confidence: 99%

Contrast-induced acute kidney injury: A review of practical points

Özkök¹,

Özkök²

2017

WJN

119

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Contrast-induced acute kidney injury (CI-AKI) is one of the most common causes of AKI in clinical practice. CI-AKI has been found to be strongly associated with morbidity and mortality of the patients. Furthermore, CI-AKI may not be always reversible and it may be associated with the development of chronic kidney disease. Pathophysiology of CI-AKI is not exactly understood and there is no consensus on the preventive strategies. CI-AKI is an active research area thus clinicians should be updated periodically about this topic. In this review, we aimed to discuss the indications of contrast-enhanced imaging, types of contrast media and their impact on nephrotoxicity, major pathophysiological mechanisms, risk factors and preventive strategies of CI-AKI and alternative non-contrast-enhanced imaging methods.

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“…Contrast-induced acute kidney injury (CI-AKI) is a common cause of renal failure associated with prolonged hospitalization, morbidity and mortality [1,2]. Classically, CI-AKI has been defined as an increase in plasma creatinine of more than 0.5 mg/dl or 25-50% of the baseline value, after administration of contrast and in the absence of other explanations for the increase [3,4].…”

Section: Introductionmentioning

confidence: 99%

Comparison of Intravenous and Oral Hydration in the Prevention of Contrast-Induced Acute Kidney Injury in Low-Risk Patients: A Randomized Trial

Martín-Moreno

Varo²,

Martı́nez-Ansó

et al. 2015

Nephron

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Aims: Contrast-induced acute kidney injury (CI-AKI) is a common cause of renal failure. We evaluated the effectiveness of oral sodium citrate versus intravenous (IV) sodium bicarbonate for CI-AKI prophylaxis as well as their influence on kidney injury biomarkers. Material and Methods: A randomized, controlled, single-center study including 130 hospitalized patients (62.3% men), who were randomized to receive sodium bicarbonate (1/6 men, 3 ml/kg/h for 1 h; n = 43), oral sodium citrate (75 ml/10 kg divided into 4 doses; n = 43) or nonspecific hydration (n = 44) before contrast administration, was conducted. Serum creatinine and kidney injury biomarkers (cystatin C, neutrophil gelatinase-associated lipocalin, interleukin-8, F2-isoprostanes and cardiotrophin-1 [CT-1]) were assessed. Results: Incidence of CI-AKI was 9.2% with no differences found between hydration groups: 7.0% in sodium bicarbonate group, 11.6% in oral sodium citrate group and 9.1% in the nonspecific hydration group. Urinary creatinine and urinary CT-1/creatinine ratio decreased 4 h after contrast infusion (p < 0.001), but none of the biomarkers assessed were affected by the treatments. Conclusions: There were no differences in hydration with oral sodium citrate and IV sodium bicarbonate for the prophylaxis of CI-AKI. Therefore, oral hydration represents a safe, inexpensive and practical method for preventing CI-AKI in low-risk patients. No effect on biomarkers for kidney injury could be demonstrated.

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Predictors of Outcomes of Contrast-Induced Acute Kidney Injury After Percutaneous Coronary Intervention in Patients With Chronic Kidney Disease

Cited by 43 publications

References 23 publications

Acute kidney injury in cirrhotic patients undergoing contrast-enhanced computed tomography

Acute kidney injury in cirrhotic patients undergoing contrast-enhanced computed tomography

Contrast-induced acute kidney injury: A review of practical points

Comparison of Intravenous and Oral Hydration in the Prevention of Contrast-Induced Acute Kidney Injury in Low-Risk Patients: A Randomized Trial

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