Postoperative Sagittal Sinus Thrombosis after Spinal Anesthesia

Schou, J; Scherb, Michael

doi:10.1213/00000539-198605000-00030

Cited by 32 publications

(22 citation statements)

References 0 publications

Supporting

Mentioning

Contrasting

Order By: Relevance

“…In addition, IVT has occurred after a variety of forms of spinal dural injury, suggesting a correlation with the dural injury itself and not with the particular manner in which the injury occurred. Thus, IVT has been reported after unintentional dural penetration during epidural anesthesia [13,[19][20][21][22] or injections [16], diagnostic lumbar puncture [8-10, 14, 15, 17, 18, 22], spinal anesthesia [13,23], intrathecal injections [11,12], and myelography [22,24,25]. Finally, when IVT occurs after spinal dural injury there is a nearly constant association with PDPH, which was present in at least 26 of the 29 reported cases, including our own.…”

Section: Discussionmentioning

confidence: 54%

Intracranial Venous Thrombosis After Placement of a Lumbar Drain

Miglis

Levine

2009

Neurocrit Care

View full text Add to dashboard Cite

show abstract

Section: Discussionmentioning

confidence: 54%

Intracranial Venous Thrombosis After Placement of a Lumbar Drain

Miglis

Levine

2009

Neurocrit Care

View full text Add to dashboard Cite

show abstract

“…penetrating or close head injury [57][58][59][60][61][62][63][64][65][66][67][68][69][70][71][72][73][74], neurosurgery [75,76], electrical injury [77,78], and lumbar puncture [79][80][81][82][83][84][85], have been linked to a higher risk of CVST.…”

Section: Mechanical Precipitants (Trauma Jugular Catheter Surgery mentioning

confidence: 99%

Risk Factors of Cerebral Vein and Sinus Thrombosis

Freitas

Bogousslavsky

2007

Handbook on Cerebral Venous Thrombosis

View full text Add to dashboard Cite

The risk factors for deep venous thrombosis (and for cerebral vein and sinus thrombosis, CVST) differ from those for arterial disease. The risk factors for venous thrombosis are linked to the Virchow triad of stasis of the blood, changes in the vessel wall, and changes in the composition of the blood, especially the first and third of these. Risk factors are usually divided into acquired (e.g. surgery, trauma, pregnancy, puerperium, lupus anticoagulant, malignant disease, and female hormones) and genetic (congenital thrombophilia). However, the separation of genetic and acquired risk factors is somewhat artificial, since they have additive effects and venous thrombosis is often multifactorial. In this review, we discuss acquired risk factors for CVST. These include hormonal changes (e.g. oral contraceptives use, hormone replacement therapy, pregnancy and puerperium), mechanical precipitants (e.g. head trauma, jugular catheterization, surgery, lumbar puncture), local and generalized infections, cancer, acquired prothrombotic states (e.g. hyperhomocysteinemia, nephrotic syndrome), inflammatory diseases (e.g. vaculitis, intestinal inflammatory disease), hematological disorders, neurological diseases (e.g. dural arteriovenous malformations, spontaneous intracranial hypotension), drugs and other situations. However, only some conditions are consistently present in case series, while many appear only in anecdotal reports. Thus, in most situations, a causal link cannot be established. Determining a cause-and-effect relationship is essential for developing preventive, diagnostic, and therapeutic strategies. Therefore, further multicentered, case-controlled studies are crucial for better understanding the pathogenesis of CVST.

show abstract

“…1 The predisposing factor includes birth control pills, pregnancy and postpartum, postoperative state, hypercoagulable conditions, trauma, paranasal infection, dehydration and medications. [3][4][5] In our case, the predisposing cause was birth control pill. The interesting matter in this patient was isolated throlard vein thrombosis without dural sinus vein thrombosis that is very rare.…”

Section: Discussionmentioning

confidence: 99%