Postoperative intestinal motility an experimental study on dogs

Abstract

MAYleft-sided predominance of iliofemoral venous thrombosis and of post-thrombotic stricture formation. SUlMlMARYThe gross anatomy of the inferior vena caval bifurcation has been studied by dissection, corrosion cast preparation, and phlebography.Results have shown that :-I . T h e right common iliac artery crossed the junction of the left and right common iliac veins in 73 of 93 dissections.2. Compression of the left common iliac vein where it is crossed by the right common iliac artery was present in 29 of 54 phlebograms and in 8 of 9 corrosion casts, but in 5 of the latter there was no diminution in cross-sectional area.3. Bands within the lumen of the left common iliac vein were present in 14 of IOO dissections, in 2 of 9 casts, and in 8 of 55 phlebograms.4. There is no evidence that bands are the result of thrombosis, but their aetiology remains in doubt. 5. Symptoms rarely, if ever, result from the presence of left common iliac compression or bands, but these may influence the localization of iliofemoral venous thrombosis and of post-thrombotic stricture formation.

Effect of circumcostal gastropexy on gastric myoelectric and motor activity in dogs

“…Opinions differ as to how long changes in myoelectrical activity persist after laparotomy and abdominal surgery. Some investigators have concluded that early postoperative intestinal motility and myoelectrical activity are normal in dogs (Baker andWebster 1968, Carmichael andothers 1977); others disagree (Smith and others 1977, Bueno and others 1978, Morris and others 1983.…”

Duodenal motility is not affected during duodenotomy in pigs

“…Others also described augmented periods, but the disordered contractile activity directly after surgery was followed by a period of quiescence (Baker and Webster, 1968). In the pig, a normal MMC length consists of 70-115 min (Rayner and Wenham, 1986), while only few data describe the immediate effects of surgery on duodenal motility.…”

LOOP ILEOSTOMY CLOSURE (review)

“…Несоответствие ширины просветов функционирующей и отключённой частей подвздошной кишок на фоне развития отёка в зоне кишечного шва приводят к образованию временного механического препятствия в зоне анастомоза. Другой причиной нарушений проходимости кишечника после закрытия петлевой илеостомы следует считать ПП ЖКТ, представляющий собой одну из главных проблем в хирургии, частота развития которого составляет 3-17% [3,5,9,12,14,16,17,19,[21][22][23]27,28,[30][31][32]55]. В основе патогенетического механизма развития ПП ЖКТ лежит множество факторов, среди которых основными являются: операционная травма, нарушение гуморальной регуляции кишечника, угнетение спинномозговых рефлексов, повышенная активность симпатической иннервации, назначение опиоидных препаратов, которые воздействуют на одноименные рецепторы, а также нарушения водно-электролитного и белкового балансов.…”