Posterior alien hand syndrome after a right thalamic infarct

Marey-López, José; Rubio-Nazabal, Eduardo; Alonso-Magdalena, L; Lopez-Facal, S

doi:10.1136/jnnp.73.4.447

Cited by 43 publications

(39 citation statements)

References 17 publications

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“…1,3,6 The primary difference between our patient and those with callosal or frontal causes is our patient's severe sensory deficit, 5 which has also been seen in thalamic alien hand syndrome. 7 The behavior of the alien hand itself was indistinguishable from cases described in the literature from lesions at other sites. 1,5 This patient's unusual clinical presentation and preserved strength delayed the diagnosis of stroke.…”

mentioning

confidence: 79%

The Stroke that Struck Back: An Unusual Alien Hand Presentation

Spector

Freeman

Cheshire

2009

Journal of Stroke and Cerebrovascular Diseases

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mentioning

confidence: 79%

The Stroke that Struck Back: An Unusual Alien Hand Presentation

Spector

Freeman

Cheshire

2009

Journal of Stroke and Cerebrovascular Diseases

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“…A number of posterior variants are reported due to thalamic infarcts [10,25,26,28,69]. It was suggested that a disconnection syndrome could be produced by a remote effect or Bdiaschisis^of thalamic stroke.…”

Section: Interhemispheric Disconnection Theorymentioning

confidence: 98%

“…Uncommonly, the occipital lobe and thalamus are involved [24]. [9,25,26] Common etiologies are neurodegeneration of the parieto-occipital cortex (corticobasal syndrome [CBS], Creutzfeldt-Jakob disease [CJD]), and stroke in the parietal lobe or posterior cerebral artery territory [27•]. Historical examples include Levine and Rinne's Bopticosensory ataxia.Ê xceptions to the classification scheme do not infrequently occur.…”

Section: Posterior Variantmentioning

confidence: 99%

Alien Hand Syndrome

Hassan

Josephs

2016

Curr Neurol Neurosci Rep

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Alien hand syndrome (AHS) is a rare disorder of involuntary limb movement together with a sense of loss of limb ownership. It most commonly affects the hand, but can occur in the leg. The anterior (frontal, callosal) and posterior variants are recognized, with distinguishing clinical features and anatomical lesions. Initial descriptions were attributed to stroke and neurosurgical operations, but neurodegenerative causes are now recognized as most common. Structural and functional imaging and clinical studies have implicated the supplementary motor area, pre-supplementary motor area, and their network connections in the frontal variant of AHS, and the inferior parietal lobule and connections in the posterior variant. Several theories are proposed to explain the pathophysiology. Herein, we review the literature to update advances in the understanding of the classification, pathophysiology, etiology, and treatment of AHS.

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“…Chronic: "thalamic dementia", artery (PTPA) artery [5] mesencephalic artery [9] Dorsal internal medullary apraxia, psychoorganic Deep interpeduncular artery [8] lamina symptoms, transcortical Ventromedial pulvinar aphasia [1,2,3,9,13,[15][16][17][18] Branches: rubral tion. Additional digital subtraction angiography (DSA) was performed in ten patients who had stenosis or occlusion of the distal vertebral artery (VA) (V3/V4-segments) and/or the basilar artery (BA) on MRA, and intraarterial thrombolytic therapy was considered in the acute stage of stroke.…”

Section: Introductionmentioning

confidence: 99%

Assessment of paramedian thalamic infarcts: MR imaging, clinical features and prognosis

et al. 2004

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Considering the highly variable vascular supply of the thalamic nuclei, MRI and clinical syndromes can be heterogeneous in ischemic diseases. We attempt to determine MRI pattern and to analyse neurological features and prognosis of paramedian infarcts. In a prospective case series within 5 years from 1999 to 2003, MRI, MRA and clinical symptoms of 38 consecutive patients were analysed. The inferomedial (posterior thalamoperforating artery) territory was affected in 89%, and lesions in the anterolateral (tuberothalamic artery) territory occurred in 42%. However, definite attribution to anterolateral or inferomedial territories was not possible in 13%. Neurological manifestations were somnolence (87%), hemisyndromes (79%), cognitive deficits (58%), oculomotor nerve palsies (53%) and vertical gaze palsies (39%). The most common aetiologies were cardiac embolism (42%), intraarterial embolism (16%), small vessel disease (13%) and large artery arteriosclerosis (13%). Pathological MRA findings were encountered in 55%, and in 18%, lesions were only visible on diffusion-weighted imaging. Correlation of MRI pattern and neurological symptoms points out anterolateral thalamic lesions as the cause of amnestic deficits. Intracranial MRA allows a non-invasive prediction of basilar tip occlusion. Our results underline the necessity of additional diffusion-weighted imaging in detecting small thalamic and midbrain lesions.

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Posterior alien hand syndrome after a right thalamic infarct

Cited by 43 publications

References 17 publications

The Stroke that Struck Back: An Unusual Alien Hand Presentation

The Stroke that Struck Back: An Unusual Alien Hand Presentation

Alien Hand Syndrome

Assessment of paramedian thalamic infarcts: MR imaging, clinical features and prognosis

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