Postencephalitic pure anomic aphasia: 2-year follow-up

Okuda, Bungo; Kawabata, Keita; Tachibana, Hideki; Sugita, Minoru; Tanaka, Harumi

doi:10.1016/s0022-510x(01)00524-x

Cited by 6 publications

(5 citation statements)

References 13 publications

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“…In terms of the cognitive operations and brain regions they affect (Fig. 1), the dynamic aphasia observed with head trauma (Luria, 1970) or cerebral tumours (Costello and Warrington, 1989) and the loss of single-word comprehension observed in temporal lobe encephalitis (Okuda et al, 2001) may be closer analogues of the degenerative aphasias than the classical transcortical aphasias of vascular disease.…”

Section: Neurobiology Of the Progressive Aphasiasmentioning

confidence: 99%

“…Furthermore, processing occurs in a distributed network of brain areas that is vulnerable to a variety of acute and chronic pathological states (Levelt, 1989; Price et al, 1993; Levelt, 2001; Blank et al, 2002; Gorno-Tempini et al, 2004). The differential diagnosis of word-finding difficulty therefore encompasses a wide spectrum of acute and chronic disorders as diverse as delirium (Geschwind, 1964), aphasic stroke (Kertesz and McCabe, 1977), encephalitis (Okuda et al, 2001), major depression (Georgieff et al, 1998) and psychosis (Critchley, 1964), head injury (Levin et al, 1976), temporal lobectomy (Langfitt and Rausch, 1996) and metabolic and genetic disorders (Spinelli et al, 1995). In particular, however, it is a leading symptom of a number of degenerative conditions: the progressive aphasias (Mesulam, 1982, Hodges et al, 1992; Mesulam, 2003; Gorno-Tempini et al, 2004).…”

Section: Introductionmentioning

confidence: 99%

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Word-finding difficulty: a clinical analysis of the progressive aphasias

Rohrer

Knight

Warren

et al. 2007

Brain

149

111

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The patient with word-finding difficulty presents a common and challenging clinical problem. The complaint of 'word-finding difficulty' covers a wide range of clinical phenomena and may signify any of a number of distinct pathophysiological processes. Although it occurs in a variety of clinical contexts, word-finding difficulty generally presents a diagnostic conundrum when it occurs as a leading or apparently isolated symptom, most often as the harbinger of degenerative disease: the progressive aphasias. Recent advances in the neurobiology of the focal, language-based dementias have transformed our understanding of these processes and the ways in which they breakdown in different diseases, but translation of this knowledge to the bedside is far from straightforward. Speech and language disturbances in the dementias present unique diagnostic and conceptual problems that are not fully captured by classical models derived from the study of vascular and other acute focal brain lesions. This has led to a reformulation of our understanding of how language is organized in the brain. In this review we seek to provide the clinical neurologist with a practical and theoretical bridge between the patient presenting with word-finding difficulty in the clinic and the evidence of the brain sciences. We delineate key illustrative speech and language syndromes in the degenerative dementias, compare these syndromes with the syndromes of acute brain damage, and indicate how the clinical syndromes relate to emerging neurolinguistic, neuroanatomical and neurobiological insights. We propose a conceptual framework for the analysis of word-finding difficulty, in order both better to define the patient's complaint and its differential diagnosis for the clinician and to identify unresolved issues as a stimulus to future work.

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Section: Neurobiology Of the Progressive Aphasiasmentioning

confidence: 99%

Section: Introductionmentioning

confidence: 99%

Word-finding difficulty: a clinical analysis of the progressive aphasias

Rohrer

Knight

Warren

et al. 2007

Brain

149

111

View full text Add to dashboard Cite

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“…Translation of thoughts and images into words is impaired. Pure anomic aphasia is likely to arise from either an inferior temporal lesion or a temporo-parietal lesion 7. Our patient has had left temporal lesion on brain MRI on admission.…”

Section: Discussionmentioning

confidence: 77%

Nominal dysphasia and euphoria caused by EBV encephalitis

Çarman¹,

Yakut

Ekici

et al. 2013

BMJ Case Reports

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“…Several studies have reported cases with anomia subsequent to meningoencephalitis caused by herpes simplex. [2][3][4][5][6][7][8] Few studies have characterized the language deficits. 5, 18 Lowe, Knapp and Lambon Ralph reported that jargon was used and there was phonological impairment, anomia and mild impairment of comprehension.…”

Section: -17mentioning

confidence: 99%

“…1 Studies have shown that meningoencephalitis early in life is a risk factor for development of psychiatric, behavioral or cognitive complications later in life Nonetheless, some neuropsychological studies have reported that anomia is one of the most frequently found language abnormalities in cases of post-herpetic meningoencephalitis. [2][3][4][5][6][7][8] Other language deficits have only rarely been described in these individuals. 5 Early diagnosis makes it possible to establish better criteria for optimal rehabilitation of linguistic and cognitive difficulties in this patient group.…”

Section: Introductionmentioning

confidence: 99%

Aphasia and herpes virus encephalitis: a case study

Soares-Ishigaki¹,

Luchesi²,

Pieri³

et al. 2012

Sao Paulo Med. J.

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CONTEXT: Meningoencephalitis early in life, of any etiology, is a risk factor for development of subsequent sequelae, which may be of physical, psychiatric, behavioral or cognitive origin. Anomia is a language abnormality frequently found in such cases, and other language deficits are rarely described. The aim of this study was to describe the cognitive and linguistic manifestations following a case of herpetic meningoencephalitis in a 13-year-old patient with eight years of schooling. CASE REPORT: The patient underwent a speech-language audiology assessment nine months after the neurological diagnosis. The battery of tests included the Montreal-Toulouse Language Assessment test protocol (MT Beta-86, modified), the description from the Cookie Theft task of the Boston Diagnostic Aphasia Examination (BDAE), an informal assessment of the patient's logical and mathematical reasoning, and the neuropsychological subtests from the WAIS-III scale, which assess working memory. The patient presented mixed aphasia, impairment of short-term memory and working memory, and dyscalculia. This case also presented severe cognitive and linguistic deficits. Prompt diagnosis is crucial, in order to enable timely treatment and rehabilitation of this neurological infection and minimize the cognitive deficits caused by the disease.

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Postencephalitic pure anomic aphasia: 2-year follow-up

Cited by 6 publications

References 13 publications

Word-finding difficulty: a clinical analysis of the progressive aphasias

Word-finding difficulty: a clinical analysis of the progressive aphasias

Nominal dysphasia and euphoria caused by EBV encephalitis

Aphasia and herpes virus encephalitis: a case study

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