Post-traumatic syringomyelia

Vernon, Jordyn; Silver, Jonathan; Ohry, A

doi:10.1038/sc.1982.64

Cited by 45 publications

(27 citation statements)

References 10 publications

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“…These two phenomena are increased when there is a blockage in the subarachnoid space. The signi®cant correlation with factors of venous hyperpressure found in this present study, the worsening reported in the literature after straining, 8,24 weightlifting exercises, 25 forced expiratory through a closed glottis, coughing or Vasalva manoeuver, 24 lithotripsy, 26 comfort this theory.…”

Section: Radiological Analysis Of the Lesion Sitesupporting

confidence: 85%

Post-traumatic syringomyelia and post-traumatic spinal canal stenosis: A direct relationship: Review of 75 patients with a spinal cord injury

Perrouin-Verbe¹,

Lenne-Aurier²,

Robert³

et al. 1998

Spinal Cord

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This study aims to demonstrate predictive factors for post traumatic syringomyelia (PTS), and in particular to correlate the role of insu ciency of reduction of a spinal fracture with the occurrence of syringomyelia. One hundred and twenty-eight spinal cord injured patients (SCI) were studied during the years 1992 and 1993. Among them, 75 underwent a complete and reliable evaluation including: review of the initial vertebral lesion, and of the surgery report, and a radiological study of the lesion site with standard X-rays, a CT scan, and an MRI. The CT Scan included slices in sagittal reconstructions and in the axial plane at the site of injury with the calculation of a percentage of canal stenosis in the two planes of the space. An MRI was carried out with T1 and T2 weighted images, including sagittal entire cord images in addition to sagittal and axial slices centred on the site of injury. A syrinx was diagnosed in 28% of the patients. The occurrence of a syrinx is signi®cantly correlated with spinal canal stenosis in the sagittal plane (DD) with a P50.001 and in the axial plane (DS) (P50.05). This present study demonstrates the major role of the insu ciency of reduction of the vertebral lesion in the genesis of a syrinx. The quality of the initial treatment of the vertebral injury is the ®rst step in the prevention of a syrinx. The treatment of a syrinx, besides techniques of drainage, must also take into account the spinal realignment.

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Section: Radiological Analysis Of the Lesion Sitesupporting

confidence: 85%

Post-traumatic syringomyelia and post-traumatic spinal canal stenosis: A direct relationship: Review of 75 patients with a spinal cord injury

Perrouin-Verbe¹,

Lenne-Aurier²,

Robert³

et al. 1998

Spinal Cord

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“…During the first few hours and days, neurological deterioration may result from edema and other secondary changes involving one or two spinal cord segments adjacent to the original site of trauma. After a period of neurological stability, late delayed complications, such as progressive post-traumatic cystic myelopathy (syringomyelia) [1][2][3] or progressive post-traumatic myelomalacic myelopathy, 4,5 may produce new ascending neurological symptoms months, years, or even decades after the original injury.…”

Section: Introductionmentioning

confidence: 99%

Subacute delayed ascending myelopathy after low spine injury: case report and evidence of a vascular mechanism

Schmidt

2005

Spinal Cord

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Study design: Case report of a patient with subacute delayed myelopathy after an acute low thoracic spine injury. Objectives: To draw awareness to a rarely described complication with potential to add devastating neurological insult to the original spinal cord injury, and to discuss evidence supporting a vascular mechanism. Setting: Health Science Centre, Winnipeg, Manitoba, Canada. Case report: A 35-year-old woman developed clinical and MRI evidence of ascending myelopathy, extending up to C5, 16 days after a T11/12 fracture dislocation. The distribution of MRI signal abnormality, MRI evidence of prominent venous markings, and association with upright mobilization and the wearing of a thoraco-lumbo-sacral orthosis, suggest that elevated spinal venous pressure in conjunction with low arterial pressure may have induced impaired spinal cord vascular perfusion. Conclusion: After recent spinal cord injury, factors exacerbating spinal venous hypertension and/or arterial hypotension may in some patients lead to impaired spinal cord perfusion. These factors should be considered and corrected if symptoms or signs of progressive myelopathy emerge in the first few days or weeks after injury.

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“…The incidence of this rather rare late complication of paraplegia is estimated in the literature to be about 1.3° 0' which is much higher than the incidence of primary syringomyelia: about 0.01 °0 (Barnett, 1966;Vernon, 1982). In a recent publication an even higher incidence was noted: 3.2%, but in the case of complete paraplegia the figures reached those noted in the literature of 1.4°0 (Rossier, 1985).…”

Section: Post-traumatic Syringomyelia: Generalitiesmentioning

confidence: 60%