“…and decalcification (dystrophy), 7,9,[20][21][22][23][24][25] and, in association with sympathetic impulse overstimulation from the same source, there is an accompanying dysfunction of the neural, vascular and visceral systems and lowering of the pain threshold. 4,7,16,[26][27][28] Investigation of antidromic impulses in afferent nerves from over-stimulation has been under investigation since 1874 by Goltz, Stickler, Bayliss, Gaskell, Langlcy, Foerster, Hinsey, Gasser, de Takats.…”