“…Some of them are associated with the arachidonic acid pathway (LTC4S [ 79 ], COX2 [ 80 ], cysLTR1 [ 81 ], cysLTR2 [ 82 ]), and some are related to eosinophilic inflammation (ACE [ 83 ], CCR3 [ 84 ], NLRP3 [ 85 ], TBX21 [ 86 ]) and pathogenesis of nasal polyps (TGF-β1 [ 87 ]). There are also gene polymorphisms whose relationship with AIA is not fully ascertained (FSIP1 [ 88 ], CEP68 [ 72 ], SLC22A2 [ 89 , 90 ], ADAM33 [ 91 ], EMID2 [ 92 ], TLR3 [ 93 ], ADORA1 [ 94 ], CACNG6 [ 95 ], WDR46 [ 96 ], ZNRD1 [ 97 ], KIFC1 [ 98 ], CYP2C19 [ 99 ]).…”