Portal and Mesenteric Vein and Inferior Vena Cava Thrombosis Associated with Antiphospholipid Syndrome.

Higa, Mariko; Kojima, Masato; Ohnuma, Shizuka; Hamanaka, Shinsuke; Yamamuro, Wataru; Sugiura, Hiroaki; Sato, Mariko

doi:10.2169/internalmedicine.40.1245

Cited by 24 publications

(8 citation statements)

References 9 publications

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“…13 It has also been reported that portal vein thrombosis often develops as a result of APS. [14][15][16] In the current case, portal hypertension seemed to be induced by portal vein thrombosis, which was possibly caused by APS that slowly developed 5 years after the onset of BCC. Irregular distribution of pulmonary circulation might be also associated with APS.…”

Section: Discussionmentioning

confidence: 61%

A case of antiphospholipid antibody syndrome that manifested in the course of basal cell carcinoma

et al. 2007

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A case of antiphospholipid antibody syndrome (APS) is presented, which manifested 5 years after onset of basal cell carcinoma (BCC). There were multiple collateral veins due to portal vein thrombosis. Because immunological abnormalities including anti-cardiolipin beta(2) glycoprotein-I antibody improved after surgical resection of BCC, it is likely that APS had occurred as a paraneoplastic syndrome with BCC. This case suggests that it is necessary to investigate the presence of APS when BCC is complicated by some coagulopathies.

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Section: Discussionmentioning

confidence: 61%

A case of antiphospholipid antibody syndrome that manifested in the course of basal cell carcinoma

et al. 2007

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“…There are several cases in the literature of great vein thrombosis (inferior vena cava, portal vein or mesenteric vein) in the presence of antiphospholipid antibodies 7,8 but superior vena cava involvement has not been described.…”

Section: Discussionmentioning

confidence: 99%

Superior vena cava syndrome – changing etiology in the third millennium

Herscovici

Szyper‐Kravitz

Altman

et al. 2011

Lupus

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Superior vena cava syndrome (SVCS), is diagnosed following different degrees of central venous system obstruction, which traditionally was caused by infections, tumors or fibrosing mediastinitis. Recently the role of SVC thrombosis secondary to indwelling central venous devices or pacemaker leads as well as different hypercoagulable states have drawn much attention. In the current review we present a 58-year-old female patient who underwent recurrent pacemaker replacements due to recurrent infections. The patient was hospitalized with superior vena cava syndrome and multiple thrombi in the upper body circulation. Additionally the evaluation was conducted for thrombophilia, which revealed the presence of high titers of antiphospholipid antibodies, suggesting the concurrent diagnosis of the antiphospholipid syndrome (APS). This case reflects the changes in the etiology of SVCS, and the need for a comprehensive evaluation of patients, in the search for additional factors that may complicate a pacemaker insertion, such as the presence of antiphospholipid antibodies. We review the relevant literature and highlight the importance for an interdisciplinary approach in the treatment of SVCS nowadays.

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“…Thrombosis of the mesenteric vein was first described by Warren and Eberhard [11] in 1935 and there have been several case reports of mesenteric vein thrombosis associated with aCL antibodies [12]. Portal and superior mesenteric [13].…”

Section: Discussionmentioning

confidence: 99%

Endoscopically Removed Hepatolithiasis Associated with Cavernous Transformation of the Portal Vein and Antiphospholipid Antibody Syndrome

et al. 2006

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Portal and Mesenteric Vein and Inferior Vena Cava Thrombosis Associated with Antiphospholipid Syndrome.

Cited by 24 publications

References 9 publications

A case of antiphospholipid antibody syndrome that manifested in the course of basal cell carcinoma

A case of antiphospholipid antibody syndrome that manifested in the course of basal cell carcinoma

Superior vena cava syndrome – changing etiology in the third millennium

Endoscopically Removed Hepatolithiasis Associated with Cavernous Transformation of the Portal Vein and Antiphospholipid Antibody Syndrome

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