Pontine hemorrhage presenting as ataxic hemiparesis.

Schnapper, Robert A.

doi:10.1161/01.str.13.4.518

Cited by 48 publications

(7 citation statements)

References 11 publications

(10 reference statements)

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“…Cerebellar symptoms homolateral to the hemiparesis have been described in a few cases of infarcts of the pons (Fisher, 1967(Fisher, , 1978Schnapper, 1982;Tu hrim et al , 1982;Kobatake and Shinohara 1983). Even if such symptoms may be overshad� owed by severe motor deficits, as in our case, the relative reduction of cerebellar perfusion observed may represent their functional correlate.…”

Section: Case Reportsupporting

confidence: 53%

Cerebellar Diaschisis in Pontine Ischemia. A Case Report with Single-Photon Emission Computerized Tomography

Perani

Lucignani

Pantano³

et al. 1987

J Cereb Blood Flow Metab

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Regional cerebral and cerebellar blood flows were studied by N, N, N'-trimethyl- N'-(2-hydroxy-3-methyl-5-[123I]iodobenzyl)-1,3-propanediamine 2 HC1 (1-123 HIPDM) and single-photon emission computerized tomography (SPECT) in a patient with an ischemic lesion of the pons. An asymmetry of perfusion of the cerebellar hemispheres, normal on transmission computerized tomography scan, was demonstrated by SPECT studies in the early acute phase and confirmed 15 days after. This finding may be related to the interruption of the corticopontocerebellar pathways.

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Section: Case Reportsupporting

confidence: 53%

Cerebellar Diaschisis in Pontine Ischemia. A Case Report with Single-Photon Emission Computerized Tomography

Perani

Lucignani

Pantano³

et al. 1987

J Cereb Blood Flow Metab

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“…Small hemorrhage as well as lacunar infarct in the basis pontis was reported to produce ataxic hemiparesis 19 2l -u and pure motor hemiparesis. 13 The 2 patients with pontine hemorrhage in this study showed ataxic hemiparesis with sensory impairment or sensorimotor stroke.…”

Section: Discussionmentioning

confidence: 99%

Lacunar syndrome due to intracerebral hemorrhage.

Mori¹,

Tabuchi²,

Yamadori³

1985

Stroke

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SUMMARY It has been recognized that small intracerebral hemorrhage not uncommonly produced lacunar syndromes. In this study, we examined cases of intracerebral hemorrhage presenting as lacunar syndromes. Of 174 cases with recent intracerebral hemorrhage, 19 presented with a lacunar syndrome: 4 presented with pure motor hemiparesis, 5, ataxic hemiparesis, 3, dysarthria-clumsy hand syndrome, 7, sensorimotor stroke, and, none, pure sensory stroke. The sites of hemorrhage were capsular in 11, putaminal in 6, and pontine in 2. In these 19 patients, 17 were hypertensive, and the signs characteristic of parenchyma! hemorrhage, e.g., gradual onset, headache, nausea, vomiting and stiff neck, were absent or very rare. Computed tomography revealed that one third of the patients had one or more non-symptomatic lacunae in the basal ganglia, the corona radiata or the anterior limb of the internal capsule. These observations suggests that hypertensive intracerebral hemorrhage causes lacunar syndrome more often than previously considered and is apt to manifest ataxic hemiparesis and sensorimotor stroke. Computed tomography is the only way of differentiating hemorrhagic "lacunar" syndrome from lacunar infarct.

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“…* Although ataxic hemiparesis has been recognized as one of the *After we reported Case 1 at the 80th Regional Meeting (Kanto district) of Japanese Society of Neurology and we prepared the paper for publication including Case 2 which we examined immediately after the Meeting, Schnapper 6 reported a case with pontine hemorrhage present ing as ataxic hemiparesis in this Journal. lacular syndromes, 7 we emphasize that small pontine hemorrhage must be considered, as well as small in farctions, tumors or demyelinating lesions, as a possi ble causative lesion of ataxic hemiparesis.…”

Section: Discussionmentioning

confidence: 99%