Platypnea-orthodeoxia syndrome occurring after a blunt chest trauma with acute tricuspid regurgitation

Hsu, Pai‐Feng; Leu, Hsing-Bang; Lu, Tse‐Min; Yu, Wen‐Chung; Hsu, Tsui‐Lieh

doi:10.1016/j.amjmed.2004.09.010

Cited by 13 publications

(7 citation statements)

References 6 publications

Supporting

Mentioning

Contrasting

Order By: Relevance

“…These functional defects can either reposition the atrial septum (aortic dilation/aneurysm, atrial septal aneurysm, intracardiac lipoma, cardiac surgeries) or redirect the blood flow from the inferior vena cava (prominent eustachian valve). Occasionally, a tricuspid regurgitant jet can also be projected directly through the interatrial communication …”

Section: Mechanisms Of Cardiac Posmentioning

confidence: 99%

“…Occasionally, a tricuspid regurgitant jet can also be projected directly through the interatrial communication. 12 The other group of functional defects act through transient reversal of the left-to-right pressure gradient. 11 These conditions elevate right atrial pressures to the point that the left-to-right pressure gradient reverses for communication, the anatomic defect is typically a pulmonary arteriovenous malformation, or severe ventilation-perfusion mismatching.…”

Section: Mechanisms Of Cardiac Posmentioning

confidence: 99%

See 1 more Smart Citation

Cardiac Platypnea‐Orthodeoxia Syndrome: An Often Unrecognized Malady

Knapper

Schultz

Das

et al. 2014

Clinical Cardiology

View full text Add to dashboard Cite

Platypnea-orthodeoxia syndrome (POS) is a rare but clinically important form of dyspnea. The syndrome is characterized by dyspnea and arterial oxygen desaturation that occurs in the upright position and improves with recumbency. In cardiac POS, an atrial septal defect or patent foramen ovale allows communication between the right-and left-sided circulations. A second defect, such as a dilated aorta, prominent eustachian valve, or pneumonectomy, then contributes to right-to-left shunting through the interatrial connection. Diagnosis is made through pulse oximetry to confirm orthodeoxia and through transesophageal echocardiography with bubble study to visualize the shunt. Although data are limited for this rare syndrome, percutaneous closure has thus far proven safe and effective.

show abstract

Section: Mechanisms Of Cardiac Posmentioning

confidence: 99%

Section: Mechanisms Of Cardiac Posmentioning

confidence: 99%

Cardiac Platypnea‐Orthodeoxia Syndrome: An Often Unrecognized Malady

Knapper

Schultz

Das

et al. 2014

Clinical Cardiology

View full text Add to dashboard Cite

show abstract

“…This repositioning or increased mobility of the inter-atrial septum giving rise to intracardiac POS can be observed in aortic aneurysm, aortic root dilatation/elongation in individuals with an ASA, or in persistent Eustachian valve or Chiari's network [9][10][11][12]. In the setting of tricuspid regurgitation, a jet of blood can be projected to the left atrium through the inter-atrial shunt [13][14][15]. The ascending aorta and inter-atrial septum can be more horizontal in the setting of osteoporosis-related kyphoscoliosis [16,17].…”

Section: Discussionmentioning

confidence: 99%

Patent Foramen Ovale With Platypnea – Orthodeoxia Syndrome: A Case Report

Mirwais

Maiwand

Altaf

et al. 2020

Cureus

View full text Add to dashboard Cite

Platypnea-orthodeoxia syndrome (POS) is an extraordinary medical condition characterized by positional dyspnea (platypnea) and arterial desaturation or hypoxemia (orthodeoxia) in the setting of an upright position. The difficulty breathing is alleviated upon lying down. It is the opposite of orthopnea and is manifested by a decrease in oxygen saturation when changing from supine to an orthostatic position. POS can have an intracardiac or an extracardiac etiology. Herein we report a case of an 87-year-old man presenting with acute on chronic dyspnea who showed promising improvement in oxygen saturation after patent foramen ovale (PFO) closure.

show abstract

“…Generally, rupture of the chordae tendineae of the tricuspid valve is primarily induced by blunt chest trauma, and previous reports have described the occurrence of tricuspid chordal rupture due to trauma. [6][7][8][9] Although other etiologies have been reported (e.g., infective endocarditis, myocardial infarction, congenital disorders, 10,11 iatrogenic complication of endomyocardial biopsy 12,13 ), only a few studies have described spontaneous tricuspid chordal rupture. 14,15 Irrespective of the cause, POS induced by rupture of the chordae tendineae of the tricuspid valve should be rapidly diagnosed to promptly proceed with the adequate surgery needed to resolve the hemodynamic compromise.…”

Section: Discussionmentioning

confidence: 99%