Plasma cyclic adenosine 3′5′-monophosphate (AMP) levels in acute myocardial infarction

Rabinowitz, Babeth; Kligerman, Miriam; Parmley, William W.

doi:10.1016/0002-9149(74)90085-x

Cited by 15 publications

(8 citation statements)

References 24 publications

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“…Increased levels are observed in hyperthyroidism, pheochromacytoma, renovascular hypertension, uremia, idiopathic edema (see Broadus 1977) as well as in severe myocardial infarction (Rabinowitz et al 1974;Strange et al 1974), sepsis (Sibbald et al 1977) and pregnancy (Zing et al 1977). Decreased levels are reported in hypothyroidism (see Broadus 1977) juvenile-onset diabetes (Goldberg et al 1977) and postpartum (Zing et al 1977).…”

Section: Resultsmentioning

confidence: 99%

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Cyclic AMP concentrations in plasma, cerebrospinal fluid and urine in patients with acute cerebral hemispheric infarction.

Tominaga

Murakami

Abe³

et al. 1981

Tohoku J. Exp. Med.

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Section: Resultsmentioning

confidence: 99%

“…differential diagnosis of pseudohypoparathyroidism (Werder et al 1978) and prognosis of myocardial infarction (Rabinowitz et al 1974; Strange et al 1974) and of head trauma (Fleischer et al 1977). …”

mentioning

confidence: 99%

Cyclic AMP concentrations in plasma, cerebrospinal fluid and urine in patients with acute cerebral hemispheric infarction.

Tominaga

Murakami

Abe³

et al. 1981

Tohoku J. Exp. Med.

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“…Myocardial infarction in humans with a marked secretion of catecholamines is associated with a rise of cyclic-AMP in venous (24) and coronary sinus blood 156 Basic Research in Cardiology, Vol. 2 (1979) (25). 2 (1979) (25).…”

Section: Discussionmentioning

confidence: 99%

“…The increase in myocardial cyclic-AMP levels following ischemia or coronary occlusion (14,(24)(25)(26) is induced by release of catecholamines within the cardiac tissue and activation of adenyl cyclase. Venous plasma cyclic AMP is increased slightly after coronary occlusion in dogs (26) as well as in patients with acute myocardial infarction (24)(25).…”

Section: Discussionmentioning

confidence: 99%

Myocardial extraction of cyclic AMP during pacing-induced angina

Kugler¹,

Nolde²

1979

Basic Res Cardiol

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The application of cyclic AMP as a marker of myocardial ischemia in humans with coronary artery disease has been investigated during pacing-induced angina. Cyclic AMP was determined by a radioimmunoassay. In 15 patients myocardial lactate extraction at rest (20 +/- 12%) converted to production levels (-30 +/- 23%) during angina (p less than 0.0005). Insignificant changes occurred in coronary venous plasma cyclic-AMP levels. The mean myocardial cyclic-AMP extraction at rest (0.3 +/- 8.7%) converted to small release values (-6.0 +/-11%) during angina (= n.s.). No significant correlation was found between myocardial lactate and cyclic-AMP uptake or release. Therefore, cyclic AMP is an insensitive marker in the evaluation of myocardial ischemia.

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“…In subarachnoid hemorrhage such a correlation was not clear, because the number of examined cases was small. Complications should be also taken into the consideration, because severe cases of myocardial infarction (Rabinowitz et al 1974) and some types of hypertension (Hamet et al, 1974) have been reported to participate in the change of the plasma cyclic AMP levels.…”

Section: Discussionmentioning

confidence: 99%