4,5]. This pigment anomaly results from a mutation in the proto-oncogene KIT at chromosome 4, or in the gene SLUG at chromosome 8; these mutations are responsible for a lack of migration and differentiation of melanoblasts during embryogenesis [6][7][8].Clinically, this syndrome is manifested by the presence, at birth, depigmented spots that may interest the face, chest, abdomen, extremities. These spots are stable and persistent with distribution symmetry [9]. A lock of white hair, often triangular in shape, is present on the front of the scalp, sometimes with the underlying forehead [10]. In 80 to 90% of cases, this frontal white lock may be the only clinical manifestation [2,11]. Eyelashes