Physiology of the human dental pulp

Hassel, H.J. Van

doi:10.1016/0030-4220(71)90258-1

Cited by 150 publications

(55 citation statements)

References 3 publications

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“…[C: resin cement; RD: root dentin.] (31). Likewise, in bonded root canals, the low compliance of the hard tissue cage renders it impossible to contract to accommodate resin polymerization shrinkage (32).…”

Section: Discussionmentioning

confidence: 99%

Does Hybridization of Intraradicular Dentin Really Improve Fiber Post Retention in Endodontically Treated Teeth?

Pirani

Chersoni

Foschi

et al. 2005

Journal of Endodontics

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Section: Discussionmentioning

confidence: 99%

Does Hybridization of Intraradicular Dentin Really Improve Fiber Post Retention in Endodontically Treated Teeth?

Pirani

Chersoni

Foschi

et al. 2005

Journal of Endodontics

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“…The resilient ground substance limits intrapulpal pressures to the site of irritation, and is not transmitted throughout the pulp space. 51 Significant pressure differences have been observed at sites only 1 to 2mm apart. 52 Pressure from the increased tissue fluid collapses the thin-walled veins and venules only in the area of the affected pulp tissue, causing a localized vascular stasis and ischaemia, resulting in local cellular death.…”

Section: Special Features Of the Pulp With Respect To Its Circulationmentioning

confidence: 97%

An overview of the dental pulp: its functions and responses to injury

Yu¹,

Abbott²

2007

Australian Dental Journal

228

158

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The dental pulp is a unique tissue and its importance in the long-term prognosis of the tooth is often ignored by clinicians. It is unique in that it resides in a rigid chamber which provides strong mechanical support and protection from the microbial rich oral environment. If this rigid shell loses its structural integrity, the pulp is under the threat of the adverse stimuli from the mouth, such as caries, cracks, fractures and open restoration margins, all of which provide pathways for micro-organisms and their toxins to enter the pulp. The pulp initially responds to irritation by becoming inflamed and, if left untreated, this will progress to pulp necrosis and infection. The inflammation will also spread to the surrounding alveolar bone and cause periapical pathosis. The magnitude of pulp-related problems should not be underestimated since their most serious consequence is oral sepsis, which can be life threatening, and hence correct diagnosis and management are essential. Clinicians must have a thorough understanding of the physiological and pathological features of the dental pulp as well as the biological consequences of treatment interventions.

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“…Fristad.) entire pulp (van Hassel, 1971;Tonder and Kvinnsland, 1983). Paired measurements from inflamed and noninflamed areas in the same pulps are shown in Fig.…”

Section: Interstitial Fluid Pressure In the Pulpmentioning

confidence: 99%

Interstitial Fluid Pressure in Normal and Inflamed Pulp

Heyeraas

Berggreen

1999

Critical Reviews in Oral Biology & Medicine

145

127

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Tissue pressure is the hydrostatic pressure in the interstitial fluid which surrounds the pulpal cells. This pressure outside the vessels is normally considerably lower than the blood pressure inside the vessels. The dental pulp has a relatively low interstitial compliance due to its enclosure between rigid dentin walls. Accordingly, even a modest increase in pulpal fluid volume will raise the tissue pressure, which may compress blood vessels, leading to ischemia and necrosis. Inflammation may lead to an increase in both interstitial fluid volume and blood volume in the low-compliant pulp and thereby increase the tissue pressure. However, the increased tissue pressure may, in turn, initiate increased lymph flow and absorption of fluid into capillaries in nearby non-inflamed tissue. Both of these latter factors will transport fluid out of the affected area and subsequently out of the tooth and consequently lower the tissue pressure. Increased tissue pressure, whether caused by increased blood volume or increased capillary filtration, will promote outward flow of fluid through exposed dentin tubules and thereby help to protect the pulp against entry of harmful substances. It seems physiologically beneficial, therefore, for the pulp to have a high tissue pressure, which promptly increases when blood flow increases due to its low compliance.

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Physiology of the human dental pulp

Cited by 150 publications

References 3 publications

Does Hybridization of Intraradicular Dentin Really Improve Fiber Post Retention in Endodontically Treated Teeth?

Does Hybridization of Intraradicular Dentin Really Improve Fiber Post Retention in Endodontically Treated Teeth?

An overview of the dental pulp: its functions and responses to injury

Interstitial Fluid Pressure in Normal and Inflamed Pulp

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