Physical medicine Electrical activity in muscle pain

Cobb, Charles R.; Vries, Herbert A. De; Urban, Robert T.; Luekens, Clause A.; Bagg, Raymond J.

doi:10.1016/0304-3959(76)90129-9

Cited by 15 publications

(28 citation statements)

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“…The recording properties of needle electrodes, which only record from a relatively small portion of the muscle, might account for these findings. A study comparing the recording ability of different electrodes found that needle electrodes failed to demonstrate muscle spasm activity induced by local intramuscular injections of hypertonic saline [3]. By contrast, this activity was easily detected by high gain surface electrode recordings.…”

Section: Introductionmentioning

confidence: 95%

The neurophysiology of myofascial pain syndrome

Rivner

2001

Current Science Inc

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This article discusses the neurophysiology of myofascial pain syndrome. The local twitch response is a characteristic finding of this condition; it is activated by snapping palpation, pressure, or needle insertion at the trigger point. It is manifested by a burst of activity in the muscle band that contains the activated trigger point. No activity is seen at other muscles bands. Data from experiments with the rabbit indicate that this is a spinal reflex, as it is abolished by transection of the motor nerve innervating the trigger point and infusion of lidocaine. Transection of the spinal cord above the level of the trigger point fails to permanently alter the trigger point response. Needle examination recordings from trigger points show low-voltage spontaneous activity and activity resembling end-plate spikes. This activity is reduced with infusion of phentolamine and local intramuscular infusions of phenoxybenzamine. There are four hypotheses to explain the findings seen at the trigger point. The first is that trigger points are found at the muscle spindle. While this theory may explain the effects of alpha-adrenergic antagonists at the trigger point, it does not fully explain the electromyographic (EMG) findings recorded at the trigger point. The second theory is that trigger points represent hyperactive end-plate regions, as the EMG activity recorded at trigger points resembles that described at the end-plate region. Other theories that either deny the existence of myofascial pain syndrome or believe it represents a focal dystonia are also discussed.

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Section: Introductionmentioning

confidence: 95%

The neurophysiology of myofascial pain syndrome

Rivner

2001

Current Science Inc

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“…9 To circumvent the problems with studies in patient populations such as differences in pain intensity, pain localization, pain duration, medication, psychological factors, etc., several studies have been carried out with experimental muscle pain and EMG recordings in healthy subjects. 1,2,7,10,11,34 In a recent experimental study it was shown that both muscle pain and sham muscle pain caused a significant increase in the resting EMG activity of the anterior temporalis muscle. 33 The results were explained by increased EMG activity of the eye muscles and the facial muscles subserving a mimic response to pain.…”

mentioning

confidence: 98%

Experimental muscle pain does not cause long-lasting increases in resting electromyographic activity

et al. 1998

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“…42 Hence, not only does the application of the treatment translate as a wave down the spine at multiple spinal levels, but the reflex activity involved in that translation also acts at multiple spinal levels. So, if the paraspinal muscles are undergoing a pain-spasm-pain cycle 43 or have enhanced gamma motor neuron sensitivity and are responsible for asymmetric loads on the spine, then the KKT will reduce the load asymmetry. 44 …”

Section: Spinal Effects Of Specific Frequency Waveforms Gamma Motor Nmentioning

confidence: 99%

Spinal Mechanisms of Pain Control

Desmoulin

Yasin²,

Chen³

2007

The Clinical Journal of Pain

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Objectives: To demonstrate initial results using Khan Kinetic Treatment (KKT) as a chronic neck pain treatment and to present pain control mechanistic theory on which the treatment is based.Methods: A self-reported functional assessment, neck pain questionnaire, and pain medication dose were used as outcome measures for 44 matched patients randomly split into 2 groups (''treatment'' and ''control''). The treatment group underwent a treatment period consisting of several individual KKT treatments, whereas the control group continued conventional therapy.Results: Compared with a control group, the treatment group lowered both their self-recorded neck pain scores (P = 0.012) and pain medication dose (P = 0.048), although current functional assessment questionnaires (range of motion, overall activity, and recreation/work activities) did not detect changes (P = 0.233, 0.311, and 0.472, respectively). Discussion:We address the theory of the pain control mechanisms of the device in detail. Although we await randomized placebo controlled trials and additional results from ongoing mechanistic studies, initial results show that KKT is potentially an effective treatment for chronic neck pain and may contribute to the reduction of pain relieving medication.

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Physical medicine Electrical activity in muscle pain

Cited by 15 publications

References 0 publications

The neurophysiology of myofascial pain syndrome

The neurophysiology of myofascial pain syndrome

Experimental muscle pain does not cause long-lasting increases in resting electromyographic activity

Spinal Mechanisms of Pain Control

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