“…Similarly, although humans rapidly methylate inorganic arsenic after absorption and often have mixtures of parent chemical and metabolites in circulation, because C. elegans lacks an arsenic methyltransferase, we were able to isolate the effect of inorganic arsenic on mitochondrial function (Luz et al, ). Despite these differences, with expanded use of worms in environmental toxicology, worm toxicokinetics have been described for a growing list of pollutants, including nanoparticles (Yang, Lin and Liao, ), cisplatin (Crone, Aschner, Schwerdtle, Karst and Bornhorst, ), chlorpyrifos (Roh, Lee and Kwon, ), and phenanthrene (Spann, Goedkoop and Traunspurger, ). In addition to species‐specific differences, experimental culture conditions are an important consideration in molecular toxicology experiments in C. elegans .…”