“…The corneal opacity is either central or peripheral, without vascularization and endothelium and DM defects result in corneal edema. Type II is frequently bilateral, but often asymmetric, with denser corneal opacification and cornea-lenticular contact with juxtaposition of the lens or cataract [12]. Other ocular anomalies include glaucoma in 50% of cases, microcornea, cornea plana, sclerocornea, chorioretinal coloboma, iris coloboma, angle and iris dysgenesis, persistent hyperplastic primary vitreous, microphthalmia, optic nerve hypoplasia and foveal hypoplasia [4,11].…”