“…The frequency of development of prolonged CSR following CAS procedures varies from 11% to 42%, and plaque characteristics and anatomic risk factors associated with such hemodynamic depression include calcified plaque, fibrous plaque, eccentric plaque, lesions involving the carotid bulb, presence of contralateral stenosis or occlusion, length of stenosis, right-sided lesions, and balloon-to-artery ratio. [12][13][14][15][16] The clinical efficacy of CEA was established in clinical trials for symptomatic and asymptomatic carotid occlusive disease. 17,18 CAS has been recommended as a less invasive but potentially equally effective treatment for carotid disease.…”