Pericardial Disease: Etiology, Pathophysiology, Clinical Recognition, and Treatment

Shabetai, Ralph

doi:10.1007/978-1-84628-715-2_71

Cited by 3 publications

(2 citation statements)

References 120 publications

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“…Diagnosis of constrictive pericarditis requires demonstration of right heart hemodynamics typical of constriction. The findings on right heart catheterization include decreased cardiac output, equalization of right and left diastolic pressures, and the characteristic square-root sign, with a steep "y" descent in the right and left ventricular diastolic pressure tracings [Shabetai 1995]. Detection of these findings can be augmented by a 500 ml volume challenge at the time of right heart catheterization.…”

Section: Discussionmentioning

confidence: 99%

Pericardiectomy: Prompt Surgical Management of Constrictive Pericarditis

Inamdar¹,

Maimaiti²,

Lijunhong³

et al. 2015

HSF

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Section: Discussionmentioning

confidence: 99%

Pericardiectomy: Prompt Surgical Management of Constrictive Pericarditis

Inamdar¹,

Maimaiti²,

Lijunhong³

et al. 2015

HSF

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“…Accumulation of fluid beyond this limit is termed pericardial effusion. [4][5][6] As a general rule, a symptomatic pericardial effusion accessible for pericardiocentesis should be extensively evaluated to determine its etiology and hemodynamic significance and could be explored as the location for local treatment. 7 For ECP, this principle should be applied.…”

Section: Basicsmentioning

confidence: 99%

Effusive-constrictive pericarditis: current perspectives

Maisch¹

2018

JVD

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Effusive-constrictive pericarditis (ECP) is defined by concurrent pericardial effusion and pericardial constriction. Whereas the pericardial cavity is typically obliterated in patients with constrictive pericarditis without any effusion, in patients with ECP the scarred pericardium not only constricts the cardiac volume but can also put pericardial fluid under increased pressure, leading to signs suggestive of cardiac tamponade. These hemodynamic features can persist even after the pericardial effusion is removed. The underlying pathogenetic process predominantly involves the visceral pericardium or epicardium. It combines visceral pericardial constriction with pericardial inflammation. Its etiology may be as variable as the causes of effusive or constrictive pericardial disease, which may be infective (e.g., bacterial or viral), malignant or autoreactive. The label idiopathic ECP should be avoided and restricted to only those cases in whom, after thorough clinical and pathological workup including PCR for microbial agents in pericardial fluid and cardiac tissue from peri-and epicardial or endomyocardial biopsies and the assessment of autoimmune processes, no definite diagnosis can be made. Since a considerable number of ECP patients have an inflammatory and reversible pericardial reaction, they can improve under causal and/or anti-inflammatory treatment. The treatment should therefore be based first on eradicating the underlying etiological factors. If such a treatment course remains ineffective, pericardiectomy including the removal of the visceral pericardium is the remaining therapeutic option.

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Péricardites aiguës et épanchements non inflammatoires du péricarde

Loire¹

2006

EMC - Cardiologie

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Pericardial Disease: Etiology, Pathophysiology, Clinical Recognition, and Treatment

Cited by 3 publications

References 120 publications

Pericardiectomy: Prompt Surgical Management of Constrictive Pericarditis

Pericardiectomy: Prompt Surgical Management of Constrictive Pericarditis

Effusive-constrictive pericarditis: current perspectives

Péricardites aiguës et épanchements non inflammatoires du péricarde

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