Paroxysmal Hemicrania-Tic Syndrome Responsive to Acetazolamide

Sanahuja, Jordi; Vázquez, Pablo; Falguera, Miquel

doi:10.1111/j.1468-2982.2005.00882.x

Cited by 9 publications

(7 citation statements)

References 13 publications

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“…The relationship between the two entities is not completely understood. It is possible that abnormalities include impaired inhibitory mechanisms that normally control afferent activity in trigeminal nucleus, as well as a hypothalamic dysfunction (6). Secondary causes of PH described in the literature include: gangliocytoma of the sella turcica, collagen vascular disease, cerebrovascular disease, Pancoast's tumour, cavernous sinus meningioma, frontal lobe tumour, intracranial hypertension, internal carotid artery aneurysm, tuber cinereum hamartoma, occipital infarction and severe orbital imflamation (7)(8)(9).…”

Section: Discussionmentioning

confidence: 99%

Paroxysmal Hemicrania-Tic and Chiari I Malformation: An Unusual Association

Monzillo¹,

Nemoto²,

Costa³

et al. 2007

Cephalalgia

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A 54 year-old female has a clinical diagnosis of chronic paroxysmal hemicrania (CPH) according to IHS-criteria (2004) three years ago. Six months after that, she developed symptoms compatible with trigeminal neuralgia (TN) involving the second and third trigeminal territories also at the left side. Cranial and cervical spine resonance magnetic images showed extensive cervical syringomyelia that included nucleus caudalis (C2 level) region and a posterior fossa Chiari I malformation without hydrocephalus. The association between CPH and TN is called chronic paroxysmal hemicrania-tic syndrome (CPH-tic). This is the first clinical description of a secondary cause of CPH-tic syndrome in the literature.

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Section: Discussionmentioning

confidence: 99%

Paroxysmal Hemicrania-Tic and Chiari I Malformation: An Unusual Association

Monzillo¹,

Nemoto²,

Costa³

et al. 2007

Cephalalgia

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“…In 2003, Boes and co‐workers presented two of their own cases and summarized all cases published before. Since then four other cases have been published including one case of possible secondary PH‐tic syndrome . This 54‐year‐old female patient developed first PH and six months later TN.…”

Section: Secondary Cluster‐tic Syndromementioning

confidence: 96%

“…The PH‐tic syndrome was first described by Hannerz in 1993 . Since then a total of 11 additional cases have been reported . In 2003, Boes and co‐workers presented two of their own cases and summarized all cases published before.…”

Section: Secondary Cluster‐tic Syndromementioning

confidence: 99%

Tics in TACs: A Step into an Avalanche? Systematic Literature Review and Conclusions

Wöber

2017

Headache

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TACs are related to tics in various ways. TN or TN-like paroxysms may co-occur with CH, PH, and HC, labeled as cluster-tic syndrome, PH-tic syndrome, and HC-tic syndrome. Such co-occurrence was not only found in the primary TACs but also in secondary headaches resembling TACs. The initial onset of TAC and tic may be simultaneous or separated by months or years. In acute attacks, tic and TAC may occur concurrently or much more often independently of each other. The term "cluster-tic syndrome" was also used in patients with a single type of pain in a twilight zone between TACs and TN fulfilling none of the relevant diagnostic criteria. Short-lasting neuralgiform headache attacks overlap with TN in terms of clinical features, imaging findings, and therapy.

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“…Sanahuja et al [19] described a case of probable PH-tic syndrome (association of PH with trigeminal neuralgia), in which both types of pain responded dramatically to a combination of acetazolamide with carbamazepine. Rossi et al reported a case of probable PH, with occipital pain location, that repeatedly responded to suboccipital steroid injection [20].…”

Section: Paroxysmal Hemicraniamentioning

confidence: 98%