Paroxysmal bundle-branch block associated with heart disease

Comeau, Wilfrid J.; Hamilton, James G.; White, Paul D.

doi:10.1016/s0002-8703(38)90243-1

Cited by 79 publications

(11 citation statements)

References 39 publications

Supporting

Mentioning

Contrasting

Unclassified

Order By: Relevance

“…Since then reports of a number of cases have been published where the disappearance and reappearance of bundle-branch block was noted (Comeau, Hamilton, and White, 1938;Vesell, 1941;Eichert, 1946;Sandberg et al, 1951;Bergquist and Hiillen, 1956;Dressler, 1959;Wallace and Laszlo, 1961;Hallhagen, 1961;Miller, 1962). Levine (1960) drew attention to the practical value of slowing the heart rate in the diagnosis of myocardial infarction in patients with left bundle-branch block and recommended it as a useful procedure.…”

Section: Discussionmentioning

confidence: 99%

“…Deep inspiration (Herrmann and Ashman, 1931;Bergquist and Hillen, 1956), carotid sinus massage (Wallace and Laszlo, 1961;Hallhagen, 1961), and resumption of the recumbent posture (Bergquist and Hiillen, 1956) have been shown to abolish bundlebranch block. Pharmacological agents that reduce the heart rate and have been successful in re-establishing normal conduction include ergotamine tartrate (Bergquist and Hallen, 1956) reserpine (Case 1), oxygen (Baker, 1930), amyl nitrite (von Mentzingen, 1934Comeau et al, 1938), calcium gluconate (Wallace and Laszlo, 1961), and molar sodium lactate (Wallace and Laszlo, 1961). The recently discovered adrenergic beta-receptor blocking compound pronethalol ("alderlin") (Black and Stephenson, 1962) is at present being investigated as a diagnostic agent.…”

Section: Discussionmentioning

confidence: 99%

“…The recently discovered adrenergic beta-receptor blocking compound pronethalol ("alderlin") (Black and Stephenson, 1962) is at present being investigated as a diagnostic agent. The control of heart failure by digitalis (Comeau et al, 1938) and thyrotoxicosis by surgery (Packard and Graybiel, 1950) may be accompanied by disappearance of bundle-branch block.…”

Section: Discussionmentioning

confidence: 99%

See 2 more Smart Citations

Bundle-Branch Block Under Voluntary Control

Bauer¹

1964

Heart

View full text Add to dashboard Cite

Section: Discussionmentioning

confidence: 99%

Section: Discussionmentioning

confidence: 99%

Section: Discussionmentioning

confidence: 99%

See 1 more Smart Citation

Bundle-Branch Block Under Voluntary Control

Bauer¹

1964

Heart

View full text Add to dashboard Cite

“…It is considered to be evidence of the presence of organic heart disease (1). Left ventricular (LV) strain with ischemia, absolute or relative, of the dilated and hypertrophied myocardium appears to be the most common background leading to the development of left bundle branch block (LBBB) (2).…”

Section: Introductionmentioning

confidence: 99%

Impact of Pulmonary Vein Isolation on Left Bundle Branch Block Following Tachycardia-induced Cardiomyopathy in a Patient with Persistent Atrial Fibrillation

et al. 2014

View full text Add to dashboard Cite

A 61-year-old man was referred to our hospital with exertional dyspnea. Electrocardiography showed atrial fibrillation (AF) with a heart rate of 116 bpm and left bundle branch block (LBBB). Chest radiography demonstrated pulmonary congestion and cardiomegaly with a cardiothoracic ratio of 57%. Transthoracic echocardiography revealed a severely reduced left ventricular systolic function (ejection fraction: 32%), suggesting tachycardia-induced cardiomyopathy (TIC) due to AF. Following treatment for congestive heart failure and complete isolation of each pulmonary vein, the LBBB disappeared, with a complete recovery of the cardiac systolic function. This report describes a case of transient reversible LBBB associated with systolic dysfunction treated with catheter ablation.

show abstract

“…Since then this condition has been dealt with in numerous clinical reports (Comeau, Hamilton, and White, 1938; Vesell, I941; Bauer, I964) and more recent attention was drawn to the phenomenon by Rosenbaum et al (I973). It was common to explain the inconsistency of left bundle-branch patterns by morphological and functional alterations, and changes in heart rate were considered a most important factor in the appearance and disappearance of blockade.…”

mentioning

confidence: 99%

Electrophysiological findings in frequency-dependent left bundle-branch block.

Neuss¹,

Thormann²,

Schlepper³

1974

Heart

View full text Add to dashboard Cite

Seven patients with frequency-depedent left bundle-branch block were studied with programmed atrial stimulation. Cardiac conduction was analysed by His bundle recordings. Atrial driving rate was gradually increased by shortening the driving cycle length by steps of Io msec from sinus rhythm up to a cycle length at which left bundle-branch block appeared. Decreasing heart rate, left bundle-branch persisted up to a cycle length which was always 8o to 170 msec longer than that found when heart rate was increased ('zone of linking'). In all patients a depression of conductivity ('postdrive depression') after a driving period of 2 minutes (ventricular rate: Ioo to I70/min) was found.Full recovery time of the left bundle-branch did not shorten in 6 patients when heart rate was accelerated.In 4 patients even an increase offull recovery time was found if basic heart rate was within the zone of linking.In 3 patients gap IIIphenomena were observed and in 2 patients a paradoxical improvement of conduction in the left bundle-branch appeared at higher rates.The underlyng mechanism of the observed phenomena might be a frequency-dependent reduction of the upstroke velocity of action potentials. Alternative mechanisms are discussed.In I913 Lewis gave an account of a 3i-year-old patient with a transient left bundle-branch block. Since then this condition has been dealt with in numerous clinical reports (Comeau, Hamilton, and White, 1938; Vesell, I941; Bauer, I964) and more recent attention was drawn to the phenomenon by Rosenbaum et al. (I973). It was common to explain the inconsistency of left bundle-branch patterns by morphological and functional alterations, and changes in heart rate were considered a most important factor in the appearance and disappearance of blockade. Alterations in heart rate had previously been induced by indirect measures only (carotid sinus pressure, Valsalva manoeuvre, amyl nitrite inhaltion, atropine medication, etc.). Using these techniques it was impossible, however, to separate the effects of alterations in heart rate from those concomitantly induced by the autonomic nervous system. It is surprising that the frequency dependence of the phenomena has not been systematically investigated by electrical stimulation. Using this method, associated with intracardiac electrography, one can arrive at a more objective evaluation regarding the frequency dependence of bundlebranch block. For example, specifically premature Received 22 February 1974. atrial depolarizations can be used to determine the refractory periods of the involved branch at different heart rates. Subjects and methodsSeven patients with intermittent left bundle-branch block were admitted to the study. All

show abstract

Paroxysmal bundle-branch block associated with heart disease

Cited by 79 publications

References 39 publications

Bundle-Branch Block Under Voluntary Control

Bundle-Branch Block Under Voluntary Control

Impact of Pulmonary Vein Isolation on Left Bundle Branch Block Following Tachycardia-induced Cardiomyopathy in a Patient with Persistent Atrial Fibrillation

Electrophysiological findings in frequency-dependent left bundle-branch block.

Contact Info

Product

Resources

About