Parkinsonian syndrome after cardiac arrest: and neurochemical changes

W, Verslegers; Crols, R.; Kerchove, M. van den; Potter, W.P. De; Appel, B; Löwenthal, A.

doi:10.1016/s0303-8467(88)80044-1

Cited by 6 publications

(2 citation statements)

References 8 publications

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“…There are no LOE P1-or LOE P2-studies that support the use of magnetic resonance imaging (MRI) to predict outcome of comatose cardiac arrest survivors. Use of MRI to predict outcome is supported by 32 studies (LOE P3 959 -963 ; LOE P4 964 -976 ; LOE P5 [977][978][979][980][981][982][983][984][985][986][987][988][989][990] ). The timing of MRI in these studies ranged from 1 day to 10 months after sustained ROSC.…”

Section: Consensus On Sciencementioning

confidence: 99%

“…There are no LOE P1-or LOE P2-studies that support the use of computed tomography (CT) imaging to predict outcome of comatose cardiac arrest survivors. Use of CT imaging is supported by 22 studies (LOE P3 992 ; LOE P4 969,984,[993][994][995][996][997][998][999][1000][1001] ; LOE P5 980,981,985,[1002][1003][1004][1005][1006] ). The timing of CT in those studies ranged from 1 hour to 20 days after sustained ROSC.…”

Section: Consensus On Sciencementioning

confidence: 99%

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Part 8: Advanced Life Support

Morrison¹,

Deakin²,

Morley³

et al. 2010

Circulation

411

169

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Section: Consensus On Sciencementioning

confidence: 99%

Section: Consensus On Sciencementioning

confidence: 99%

Part 8: Advanced Life Support

Morrison¹,

Deakin²,

Morley³

et al. 2010

Circulation

411

169

View full text Add to dashboard Cite

Hypoxic‐ischemic damage of the basal ganglia case reports and a review of the literature

1990

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Three cases of movement disorders secondary to hypoxic-ischemic encephalopathy are reported. Despite similarities among the clinical events, the neurological syndromes produced were dissimilar. Cerebral hypoxia-ischemia typically produces lesions of the globus pallidus that may result in an akinetic rigid syndrome. Due to its unique blood supply, vascular insufficiency is found to be a major factor. Lesions in the putamen also occur, and these tend to be associated with dystonia. Recent evidence supports a specific neuronal sensitivity in the striatum, possibly due to afferent excitatory amino acid connections. These two components and changes in the levels of neurotransmitters during hypoxia-ischemia may interact to produce varied clinical outcomes. These factors must also be considered when planning therapeutic interventions.

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