Parkinsonian syndrome after acute carbon monoxidepoisoning

Gallerani, Massimo; Cecilia, Olga la; Serra, Alessia; Kuwornu, Hélè Afi; Bressan, Sabrina; Regoli, François; Manfredini, Roberto

doi:10.1053/ajem.2000.9286

Cited by 4 publications

(1 citation statement)

References 17 publications

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“…Alternatively, CO-related basal ganglia lesions including the substantia nigra lead to parkinsonism with severe akinesia without tremor or rigidity (Kawanami et al, 1998). Delayed-onset parkinsonism associated with basal ganglia lesions can occur (Gallerani et al, 2000;Pavese et al, 1999;Sohn et al, 2000). Movement disorders and concomitant cognitive impairments may develop following CO poisoning.…”

Section: Parkinsonian Syndromementioning

confidence: 99%

Neuroimaging, Cognitive, and Neurobehavioral Outcomes Following Carbon Monoxide Poisoning

Hopkins

Woon

2006

Behavioral and Cognitive Neuroscience Reviews

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Carbon monoxide is a colorless, odorless gas produced as a byproduct of combustion. Carbon monoxide is the leading cause of poisoning injury and death worldwide. Morbidity following CO poisoning includes neurologic sequelae, neuropathologic abnormalities on brain imaging, neurobehavioral changes, and cognitive impairments. It is estimated that as high as 50% of individuals with carbon monoxide poisoning will develop neurologic, neurobehavioral, or cognitive sequelae. Carbon monoxide related cognitive impairments included impaired memory, attention, executive function, motor, visual spatial, and slow mental processing speed. Given the high rate of brain related morbidity and the fact that the majority of carbon monoxide is avoidable, awareness and prevention of carbon monoxide poisoning is warranted.

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