Papilledema in the Guillain-Barré Syndrome

Drew, Arthur L.

doi:10.1001/archneurpsyc.1951.02320120077010

Cited by 27 publications

(4 citation statements)

References 16 publications

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“…In the reported cases of GBS with papilledema, fundus examinations were typically normal at the time of symptom onset, with papilledema developing days to weeks later in the middle of a protracted course. 4,5 Onset of papilledema in our patient, afflicted with an autoimmune demyelinating polyneuropathy related to GBS, appears to have been similarly delayed. This suggests that the timing…”

Section: Papilledema In An Otherwise Clinically Typical Case Of Millementioning

confidence: 55%

Papilledema in an otherwise clinically typical case of Miller Fisher syndrome

Bruce

McClelland

Stavern

2015

Canadian Journal of Ophthalmology

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Section: Papilledema In An Otherwise Clinically Typical Case Of Millementioning

confidence: 55%

Papilledema in an otherwise clinically typical case of Miller Fisher syndrome

Bruce

McClelland

Stavern

2015

Canadian Journal of Ophthalmology

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“…Although papilloedema is a well-known association of GBS,7–9 it is often under-recognised and may result in permanent vision loss when severe. It generally improves with improvement in the underlying neurologic deficits associated with GBS, as demonstrated in our case.…”

Section: Discussionmentioning

confidence: 99%

Severe papilloedema with vision loss secondary to Guillain-Barré syndrome

Moloney,

Ong,

Irani

2024

BMJ Case Rep

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We discuss a patient who presented with bilateral VI and VII cranial nerve palsies, symmetric upper and lower limb weakness and areflexia, 2 weeks following an flu-like illness. At presentation, there was no papilloedema, and her visual function was normal. Cerebrospinal fluid analysis and electrophysiology supported the diagnosis of Guillain-Barré Syndrome (GBS). She received intravenous immunoglobulins. She subsequently developed headaches and vision loss. Funduscopy demonstrated severe papilloedema with visual acuity of 6/18 right eye, 6/12 left eye with bitemporal visual field depression. Lumbar puncture revealed elevated opening pressure with high protein and normal cell count. She received acetazolamide. There was resolution of papilloedema and normal visual function at 3 months. Of note, the patient’s body mass index was 17 kg/m2.Our case highlights the rare occurrence of papilloedema in GBS, reiterating the importance of performing funduscopy on patients with any neurological diagnosis. Early detection and prompt management of papilloedema can prevent permanent vision loss.

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“…The observations so far reported in the literature on the subject, and the hypotheses and theories produced by the various authors, are summarised in the paper by GLASAUER (1964) and in the more recent ones by DARDENNE (1967) and ARSENI and MARETSIS (1967). The most accredited hypotheses may be briefly summarised as follows: the block of the C. S. F. circulation -caused by spinal tumor -with increase of the C. S. F. protein content is said to be cause of the increased intracranial pressure and papilloedema by LOVE et al (1951), while according to others (DREW and MAGEE 1951 ;JENTZER et al 1952) the increase of the C. S. F. protein content only, due to the presence of the tumor, might be sufficient (leptomeningeal reaction, toxic effect etc. ).…”

Section: Discussionmentioning

confidence: 99%