Papilledema and Idiopathic Intracranial Hypertension

Buchheit, William A.; Burton, Charles; Haag, Burritt; Shaw, David

doi:10.1056/nejm196904242801707

Cited by 92 publications

(42 citation statements)

References 48 publications

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“…The clinical syndrome was later described as "pseudotumor cerebri" by Nonne in 1904 49 and then "benign intracranial hypertension" by Foley in 1955. 10,49,50 The inclusion of "benign" in the description was challenged in 1969 by Buchheit et al, 49,51 who recognized that papilledema was present in almost 95% of the patients, which, if left untreated, could progress to optic atrophy and irreversible blindness. The term "idiopathic intracranial hypertension" was introduced in 2011 50 and may be increasingly used because the etiology and pathophysiology are poorly understood.…”

Section: Discussionmentioning

confidence: 99%

Meta-Analysis of CSF Diversion Procedures and Dural Venous Sinus Stenting in the Setting of Medically Refractory Idiopathic Intracranial Hypertension

Satti

Leishangthem

Chaudry

2015

AJNR Am J Neuroradiol

184

185

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BACKGROUND AND PURPOSE:In medically refractory idiopathic intracranial hypertension, optic nerve sheath fenestration or CSF shunting is considered the next line of management. Venous sinus stenosis has been increasingly recognized as a treatable cause of elevated intracranial pressure in a subset of patients. In this article, we present the results of the largest meta-analysis of optic nerve sheath fenestration, CSF shunting, and dural venous sinus stenting. This is the only article that compares these procedures, to our knowledge.

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Section: Discussionmentioning

confidence: 99%

Meta-Analysis of CSF Diversion Procedures and Dural Venous Sinus Stenting in the Setting of Medically Refractory Idiopathic Intracranial Hypertension

Satti

Leishangthem

Chaudry

2015

AJNR Am J Neuroradiol

184

185

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“…A thorough neurological investigation and contrast-enhanced neuroimaging is essential to rule out an intracranial mass lesion or an inflammatory process. Although various factors are involved in the pathophysiology of idiopathic intracranial hypertension (IIH) [9, 10], the majority of cases are idiopathic. IIH affects about 1–2/100,000 in the general population, but 19–21/100,000 of obese women of reproductive age [11].…”

Section: Discussionmentioning

confidence: 99%

Pseudotumor cerebri in a Patient with Goldenhar’s and Duane’s Syndromes

Tillman

Kaiser²,

Killer

2002

Ophthalmologica

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A 4-year-old boy presented with Goldenhar’s syndrome, Duane’s syndrome and bilateral papilledema. Magnetic resonance imaging of the brain was unremarkable. On lumbar puncture, the cerebrospinal fluid (CSF) pressure measured 36 cm H₂O. CSF examination was normal. The diagnosis of pseudotumor cerebri was made, and treatment with acetazolamide was started. As the papilledema did not resolve, steroids were added to the treatment. Lumbar puncture was repeated after 1 month, and pressure was found to be 30 cm H₂O. Because medical treatment was not effective in lowering the CSF pressure, optic nerve sheath fenestration was performed. Papilledema resolved over the next 2 months. To the best of our knowledge, this is the first case of Goldenhar’s syndrome associated with pseudotumor cerebri.

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“…A síndrome de HICb 22 , também chamada de meningite serosa 23 , síndrome pseudotumoral 24 , hidrocefalia otítica 25 , hidropia meningea hipertensiva 26 , hidrocefalia tóxica 27 ou hipertensão intracraniana benigna idiopática 28 , tem sido atribuída tanto ao aumento do volume sanguíneo cerebral, quanto ao edema cerebral difuso, ao aumento no volume do LCR ou à diminuição de sua absorção 29 . Ainda que se conheçam os vários agentes desencadeantes dessa entidade clínica 29,30 , continuam obscuros os mecanismos básicos de sua fisiopatogenia.…”

Section: Comentáriosunclassified

Edema cerebral crônico na neurocisticercose

Agapejev

Yela²,

Gomes³

1998

Arq. Neuro-Psiquiatr.

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RESUMO -Neste estudo retrospectivo, relatam-se as características clínicas do edema cerebral crônico (ECCr) em 34 pacientes com neurocisticercose (NCC), que apresentavam edema cerebral difuso, à tomografia computadorizada (TC), como característica comum. Todos foram tratados com dextroclorofeniramina e, 32 deles, com albendazol. O ECCr predominou no sexo feminino (73,5%) na faixa etária dos 11 -40 anos (92,3%). A cefaléia ocorreu em 94,1% dos pacientes, náuseas/vômitos em 47,1%, crises epilépticas em 41,1% e distúrbios psíquicos em 38,2%. A hiperreflexia ocorreu em 82,3% e o papiledema em 58,8% e o exame neurológico normal em 11,8%. Na TC, o edema esteve associado a calcificações em 61,8% dos casos. As pressões liquóricas foram mais elevadas (p< 0,05) antes do tratamento. Atualmente, estão assintomáticos, ou com melhora clínica, 79,4% dos pacientes (57,1% deles sem medicação). Discute-se a possibilidade do ECCr, na NCC, ser uma manifestação antigênica, sem a presença concomitante de cistos parasitários, e poder representar mais uma condição clínica associada à hipertensão intracraniana benigna.PALAVRAS-CHAVE: neurocisticercose, edema cerebral crônico, hipertensão intracraniana benigna, tratamento. Chronic brain oedema in neurocysticercosisABSTRACT -In a retrospective study, we report the clinical characteristics of chronic brain oedema (CBO) in 34 patients with neurocysticercosis (NCC) who presented diffuse brain oedema upon computed tomography (CT) as a common point. All patients received dextrochlorpheniramine, and, 94.1% of them also albendazole. A predominance of females (73.5%) aged 11-40 years (92.3%) was seen. Headache occurred in 94.1% of patients, nausea/vomiting in 47.1%, epileptic manifestations in 41.1% and psychiatric disorders in 38.2%. Hyperreflexia was obtained in 82.3% of patients, papilloedema in 58.8% and normal neurological examination in 11.8%. The CT scan of the brain showed an association of oedema with calcifications in 61.8% of cases. The cerebrospinal fluid (CSF) pressure was higher before than after treatment (p < 0.05). At the last visit, 79.4% of patients were currently asymptomatic (57.1% of them without medication). It is pointed out that CBO may be an antigenic manifestation in NCC without the concomitant presence of parasitic cysts and may represent an additional clinical condition associated with benign intracranial hypertension.KEY WORDS: neurocysticercosis, brain oedema, benign intracranial hypertension, treatment.A neurocisticercose (NCC) é a doença parasitária mais comum do sistema nervoso central (SNC), causada pela forma larvária da Taenia solium. Os sinais e sintomas decorrentes dessa infecção dependem, fundamentalmente, da resposta imunológica do hospedeiro 1-3 e, como resultado, mostram variabilidade em suas exteriorizações clínicas.As manifestações desta neuroparasitose ocorrem através de múltiplas facetas, variando desde simples enxaqueca 4 aos mais graves sinais e sintomas de hipertensão intracraniana 5 , passando por

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Papilledema and Idiopathic Intracranial Hypertension

Cited by 92 publications

References 48 publications

Meta-Analysis of CSF Diversion Procedures and Dural Venous Sinus Stenting in the Setting of Medically Refractory Idiopathic Intracranial Hypertension

Meta-Analysis of CSF Diversion Procedures and Dural Venous Sinus Stenting in the Setting of Medically Refractory Idiopathic Intracranial Hypertension

Pseudotumor cerebri in a Patient with Goldenhar’s and Duane’s Syndromes

Edema cerebral crônico na neurocisticercose

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