Palinopsia with LPDs-plus heralding a posterior–cerebral–artery ischemic stroke: the hidden face of occipital lobe seizures

A novel case of Bálint syndrome with hallucinatory palinopsia following hypoglycemic coma: opening new doors to the origin of palinopsia

“…When it is secondary to structural disease, it usually appears in patients with impaired vision who are not entirely blind and transiently during the progression or resolution of visual field defect and usually disappears in a few days. 8 , 9 According to the Bayesian theory, which states that surrounding images can be predicted using stored memories of previous images, palinopsia could be due to a mismatch in the brain’s prediction phase. 8 The pathophysiology of palinopsia is unknown, but one hypothesis suggests a local irritation of the parieto-occipital cortex by surrounding edema, 8 , 9 like the cytotoxic edema following hypoglycemic coma as occurred in our case.…”

“… 8 , 9 According to the Bayesian theory, which states that surrounding images can be predicted using stored memories of previous images, palinopsia could be due to a mismatch in the brain’s prediction phase. 8 The pathophysiology of palinopsia is unknown, but one hypothesis suggests a local irritation of the parieto-occipital cortex by surrounding edema, 8 , 9 like the cytotoxic edema following hypoglycemic coma as occurred in our case. Hallucinatory palinopsia, encompassing categorical incorporation, scene preservation, and formed image preservation, indicates visual memory encoding, processing, or retrieval dysfunction.…”