Painful Ophthalmoplegia: Oculomotor Nerve Palsy Without Mydriasis Due to Compression by Aneurysm

Roman-Campos, Gustavo; Edwards, Keith R.

doi:10.1111/j.1526-4610.1979.hed1901043.x

Cited by 14 publications

(2 citation statements)

References 5 publications

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“…In this case several factors support a diagnosis of ophthalmoplegic migraine: (1) patient's young age, (2) a positive family history of common migraine, (3) a subsequent history of repeated migrainous headache attacks, (4) the occurrence of internal ocular palsy after a prolonged headache attack (this is the usual temporal pattern of ophthalmoplegic migraine), (5) CT scan and angiography negative. We consider this case interesting because of the presence of a pure internal ophthalmoplegia without any external involvement.…”

Section: Discussionmentioning

confidence: 83%

Internal Ophthalmoplegia in Complicated Migraine: A Case Report

Bussone¹,

Mantia²,

Grazzi³

et al. 1987

Headache

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SYNOPSIS A 25 year old woman with a history of migraine came to our observation complaining of blurred vision and mydriasis in the right eye, starting several hours after a typical attack of common migraine had resolved. We believe this to be a case of ophthalmoplegic migraine characterized by only an isolated internal ophthalmoplegia without external third nerve involvement. Differential diagnosis and possible pathogenetic aspects are discussed.

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Section: Discussionmentioning

confidence: 83%

Internal Ophthalmoplegia in Complicated Migraine: A Case Report

Bussone¹,

Mantia²,

Grazzi³

et al. 1987

Headache

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“…[184][185][186] Rarely, isolated pupillary-sparing oculomotor nerve palsies may be secondary to compressive lesions, although the majority of these cases have incomplete palsies. [1][2][3]95,117,160,179,[187][188][189][190][191][192][193][194]…”

Section: Acquiredmentioning

confidence: 99%

Third Nerve Palsies

Biousse¹,

Newman²

2000

Semin Neurol

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The diagnosis and management of third nerve dysfunction varies according to the age of the patient, characteristics of the third nerve palsy, and presence of associated symptoms and signs. Indeed, third nerve palsies may be partial or complete, congenital or acquired, isolated or accompanied by signs of more extensive neurological involvement. They can result from lesions located anywhere from the oculomotor nucleus to the termination of the third nerve in the extra-ocular muscles within the orbit. Recent advances in noninvasive neuroimaging facilitate early diagnosis; however, management of a patient presenting with an isolated third nerve palsy remains a challenge.

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Pupil‐sparing oculomotor palsies with internal carotid—posterior communicating artery aneurysms

Kissel

Burde

Klingele

et al. 1983

Annals of Neurology

167

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The charts of 84 patients admitted to the Neurosurgery Service of the Washington University Medical Center between January, 1960, and July, 1981, with aneurysms at or near the junction of the internal carotid and posterior communicating arteries (ICA-PoCA) were reviewed. Special emphasis was placed on the pupillary size and reactivity of 51 patients with oculomotor nerve involvement. Seven initially had normal pupils, representing 8% of the total group and 14% of those presenting with oculomotor palsies. In 4 of these patients pupillary involvement developed within 5 days, and in 1 pupillary involvement developed in 4 months. Pupillary sparing appears to be more common than previously appreciated in patients with ICA-PoCA aneurysms and oculomotor nerve involvement. Patients with acute somatic oculomotor paresis should be observed closely for at least one week for the development of pupillary involvement. Arteriography may be indicated more frequently than previously recommended.

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Painful Ophthalmoplegia: Oculomotor Nerve Palsy Without Mydriasis Due to Compression by Aneurysm

Cited by 14 publications

References 5 publications

Internal Ophthalmoplegia in Complicated Migraine: A Case Report

Internal Ophthalmoplegia in Complicated Migraine: A Case Report

Third Nerve Palsies

Pupil‐sparing oculomotor palsies with internal carotid—posterior communicating artery aneurysms

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