Oxalate Synthesis, Transport and the Hyperoxaluric Syndromes

Williams, Hibbard E.; Wandzilak, T. R.

doi:10.1016/s0022-5347(17)40999-2

Cited by 186 publications

(91 citation statements)

References 49 publications

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“…About 75% of all kidney stones are composed primarily of calcium oxalate (Williams & Wandzilak, 1989) and hyperoxaluria is a primary risk factor for this disorder (Goldfarb, 1988;Robertson & Hughes, 1993). Urinary oxalate originates from a combination of absorbed dietary oxalate and endogenous formation from oxalate precursors such as ascorbic acid and glyoxylate (Williams & Wandzilak, 1989).…”

Section: Discussionmentioning

confidence: 99%

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Oxalate Content of Egyptian Grown Fruits and Vegetables and Daily Common Herbs

Abdel-Moemin¹

2014

JFR

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Egyptian dieticians typically rely on foreign databases to find out oxalate content of food due to unavailability of local databases. The soil, fertilizers, climate and cultivars are often very different. Therefore, the purpose of this study is to establish a local database of oxalate content in Egyptian grown fruits and vegetables and selected daily common herbs. The current study analysed the total and the soluble oxalate in 37 Egyptian grown fruits, vegetables and 9 commonly used herbs. Two methods were used for screening the Egyptian foods for oxalate concentration; the first method was AOAC 1999 and the second was enzymatic method.

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Section: Discussionmentioning

confidence: 99%

“…Urinary oxalate originates from a combination of absorbed dietary oxalate and endogenous formation from oxalate precursors such as ascorbic acid and glyoxylate (Williams & Wandzilak, 1989). Restriction of dietary oxalate intake has been proposed to prevent the formation of calcium oxalate kidney stones.…”

Section: Discussionmentioning

confidence: 99%

Oxalate Content of Egyptian Grown Fruits and Vegetables and Daily Common Herbs

Abdel-Moemin¹

2014

JFR

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“…Accumulation of oxalate in leafy plants such as spinach and Amaranthaceae leads to nutritional stress, as these plants lack the ability to catabolize oxalate. Excess oxalate in the diet of humans may lead to hyperoxaluria which has been implicated in a number of pathological conditions such as formation of calcium oxalate stones in the kidney (urolithiasis), renal failure, cardiomyopathy, and cardiac conductance disorders (Williams & Wandzilak, 1989). Oxalate decarboxylase catalyzes the decarboxylation of oxalic acid to yield formic acid and carbon dioxide (Equation 5).…”

Section: Oxalate Decarboxylase (Pdb: 1uw8)mentioning

confidence: 99%

Modeling the Metal Binding Site in Cupin Proteins

Chavez¹,

Banerjee²,

Sljivic³

2011

On Biomimetics

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“…In man, oxalate is a normal constituent of human urine, being excreted as a useless end-product of intermediary metabolism or from dietary sources [8]. Its insoluble salt of calcium is the main component of kidney stones [11]. Therefore, it is tempting to postulate that the production of kidney stones or urolithiasis in some melioidosis patients may be attributable to chronic lowlevel exposure to oxalate-producing B. pseudomallei.…”

Section: Production Of Oxalate In the Culture Supernate Of Burkholdermentioning

confidence: 99%